A new role for the Krüppel-like transcription factor KLF6 as an inhibitor of c-Jun proto-oncoprotein function (Q57908318)

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Nicolás P Koritschoner

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Claudio C Prieto

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Fernando J López-Díaz

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Bruno Chatton

https://scigraph.springernature.com/pub.10.1038/sj.onc.1208020

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language of work or name

English

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publication date

20 September 2004

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published in

Oncogene

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volume

23

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issue

50

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page(s)

8196-8205

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exact match

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cites work

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AP-1 function and regulation

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Transcriptional activation of transforming growth factor beta1 and its receptors by the Kruppel-like factor Zf9/core promoter-binding protein and Sp1. Potential mechanisms for autocrine fibrogenesis in response to injury.

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Cloning the cDNA for a new human zinc finger protein defines a group of closely related Krüppel-like transcription factors

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Analysis of human prostate cancers and cell lines for mutations in the TP53 and KLF6 tumour suppressor genes

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The Krüppel-like core promoter binding protein gene is primarily expressed in placenta during mouse development.

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Transcriptional activation of the human inducible nitric-oxide synthase promoter by Kruppel-like factor 6.

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The gut-enriched Kruppel-like factor (Kruppel-like factor 4) mediates the transactivating effect of p53 on the p21WAF1/Cip1 promoter

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Cell-specific transcription of leukotriene C(4) synthase involves a Kruppel-like transcription factor and Sp1.

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Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor

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7 January 2021

Signal Transduction and the Control of Gene Expression

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JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

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Suppression of glioblastoma tumorigenicity by the Kruppel-like transcription factor KLF6.

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Initial sequencing and analysis of the human genome

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KLF6, a candidate tumor suppressor gene mutated in prostate cancer

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Kruppel-like factor 6 (KLF6) is a tumor-suppressor gene frequently inactivated in colorectal cancer.

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Role of gut-enriched Krüppel-like factor in colonic cell growth and differentiation

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The c-Ha-ras oncogene and a tumor promoter activate the polyoma virus enhancer

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The yeast UASG is a transcriptional enhancer in human HeLa cells in the presence of the GAL4 trans-activator

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