TAL1/SCL is downregulated upon histone deacetylase inhibition in T-cell acute lymphoblastic leukemia cells (Q58042312)

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3 February 2020

title

TAL1/SCL is downregulated upon histone deacetylase inhibition in T-cell acute lymphoblastic leukemia cells (English)

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3 February 2020

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lymphoblastic leukemia

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T-cell acute lymphoblastic leukemia

inferred from title

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1

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3 February 2020

Sérgio F de Almeida

2

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3 February 2020

Maria do Carmo Fonseca

4

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3 February 2020

João T. Barata

7

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3 February 2020

author name string

A B A Laranjeira

3

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3 February 2020

J A Yunes

5

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3 February 2020

P J Coffer

6

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publication date

7 June 2011

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3 February 2020

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3 February 2020

volume

25

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issue

10

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3 February 2020

page(s)

1578-1586

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Gene expression signatures define novel oncogenic pathways in T cell acute lymphoblastic leukemia

3 February 2020

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TAL1/SCL induces leukemia by inhibiting the transcriptional activity of E47/HEB.

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Formation of in vivo complexes between the TAL1 and E2A polypeptides of leukemic T cells

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TAL1/SCL relieves the E2-2-mediated repression of VEGFR2 promoter activity.

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Preferred sequences for DNA recognition by the TAL1 helix-loop-helix proteins

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mSin3A regulates murine erythroleukemia cell differentiation through association with the TAL1 (or SCL) transcription factor

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Tal1/SCL binding to pericentromeric DNA represses transcription.

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Histone deacetylase inhibitors suppress the induction of c-Jun and its target genes including COX-2.

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PTEN posttranslational inactivation and hyperactivation of the PI3K/Akt pathway sustain primary T cell leukemia viability

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