Angiotensin II-mediated GFR decline in subtotal nephrectomy is due to acid retention associated with reduced GFR (Q58134261)

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Dietary protein causes a decline in the glomerular filtration rate of the remnant kidney mediated by metabolic acidosis and endothelin receptors.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Control of glomerular hypertension limits glomerular injury in rats with reduced renal mass

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Effect of blood pressure lowering and antihypertensive drug class on progression of hypertensive kidney disease: results from the AASK trial

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Angiotensin II receptors mediate increased distal nephron acidification caused by acid retention

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Daily oral sodium bicarbonate preserves glomerular filtration rate by slowing its decline in early hypertensive nephropathy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Increased tissue acid mediates a progressive decline in the glomerular filtration rate of animals with reduced nephron mass.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Acid retention during kidney failure induces endothelin and aldosterone production which lead to progressive GFR decline, a situation ameliorated by alkali diet.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Differences in AT2 -receptor stimulation between AT1 -receptor blockers valsartan and losartan quantified by renal interstitial fluid cGMP.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Regression of glomerulosclerosis in subtotally nephrectomized rats: effects of monotherapy with losartan, spironolactone, and their combination.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Regulation of renal 12(S)-hydroxyeicosatetraenoic acid in diabetes by angiotensin AT1 and AT2 receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Local renal aldosterone system and its regulation by salt, diabetes, and angiotensin II type 1 receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Reduced extracellular pH increases endothelin-1 secretion by human renal microvascular endothelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Intra-renal angiotensin II/AT1 receptor, oxidative stress, inflammation, and progressive injury in renal mass reduction

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

"Strict" blood pressure control and progression of renal disease in hypertensive nephrosclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Influence of nutrition on acid-base balance--metabolic aspects

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Rat renal interstitial bradykinin, prostaglandin E2, and cyclic guanosine 3',5'-monophosphate. Effects of altered sodium intake

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Increased urinary excretion of angiotensinogen is associated with risk of chronic kidney disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Urinary excretion of angiotensinogen reflects intrarenal angiotensinogen production

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Treatment of metabolic acidosis in patients with stage 3 chronic kidney disease with fruits and vegetables or oral bicarbonate reduces urine angiotensinogen and preserves glomerular filtration rate

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Bicarbonate supplementation slows progression of CKD and improves nutritional status

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388

Amelioration of metabolic acidosis in patients with low GFR reduced kidney endothelin production and kidney injury, and better preserved GFR.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFU388