A prospective study to evaluate the impact of31P‐MRS to determinate mitochondrial dysfunction in skeletal muscle of ALS patients (Q58145730)

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article by Torsten Grehl et al published January 2007 in Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders

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English	A prospective study to evaluate the impact of31P‐MRS to determinate mitochondrial dysfunction in skeletal muscle of ALS patients	article by Torsten Grehl et al published January 2007 in Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders

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scholarly article

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A prospective study to evaluate the impact of31P‐MRS to determinate mitochondrial dysfunction in skeletal muscle of ALS patients (English)

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amyotrophic lateral sclerosis

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based on heuristic

inferred from title

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based on heuristic

inferred from title

3

object named as

Klaus Müller

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author name string

Torsten Grehl

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Stephan Fischer

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Jean‐Pierre Malin

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Jochen Zange

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publication date

January 2007

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Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders

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8

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1

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4-8

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The Golgi apparatus of spinal cord motor neurons in transgenic mice expressing mutant Cu,Zn superoxide dismutase becomes fragmented in early, preclinical stages of the disease

1 reference

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Cytochrome c oxidase subunit I microdeletion in a patient with motor neuron disease.

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Aberrant copper chemistry as a major mediator of oxidative stress in a human cellular model of amyotrophic lateral sclerosis

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Ca2+ release from mitochondria induced by prooxidants

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Mitochondrial involvement in amyotrophic lateral sclerosis

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Neuroprotective effects of creatine in a transgenic animal model of amyotrophic lateral sclerosis.

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Amyotrophic lateral sclerosis: oxidative energy metabolism and calcium homeostasis in peripheral blood lymphocytes

1 reference

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Visualization of defective mitochondrial function in skeletal muscle fibers of patients with sporadic amyotrophic lateral sclerosis

1 reference

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Mitochondrial changes in skeletal muscle in amyotrophic lateral sclerosis and other neurogenic atrophies.

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Mitochondrial impairment in patients and asymptomatic mutation carriers of Huntington's disease

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Transgenic ALS mice show increased vulnerability to the mitochondrial toxins MPTP and 3-nitropropionic acid.

1 reference

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Mitochondrial DNA and respiratory chain function in spinal cords of ALS patients

1 reference

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Mitochondrial respiratory chain function in skeletal muscle of ALS patients.

1 reference

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Selective changes in mitochondria respiratory properties in oxidative or glycolytic muscle fibers isolated from G93AhumanSOD1 transgenic mice

1 reference

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Impaired oxidative metabolism in exercising muscle from ALS patients

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Mitochondrial impairment of human muscle in Friedreich ataxia in vivo

1 reference

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31P NMR examination of two patients with NADH-CoQ reductase deficiency

1 reference

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21 January 2018

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10.1080/17482960600765065

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