Congestive heart failure induces endothelial cell apoptosis: protective role of carvedilol (Q58150267)

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3 November 2019

title

Congestive heart failure induces endothelial cell apoptosis: protective role of carvedilol (English)

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3 November 2019

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congestive heart failure

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2

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3 November 2019

Ziad Mallat

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S Dimmeler

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3 November 2019

author name string

L Rössig

1

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3 November 2019

B Hugel

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3 November 2019

J M Freyssinet

5

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A Tedgui

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3 November 2019

A M Zeiher

8

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3 November 2019

language of work or name

English

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publication date

1 December 2000

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Journal of the American College of Cardiology

3 November 2019

published in

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3 November 2019

volume

36

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issue

7

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3 November 2019

page(s)

2081-2089

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Alterations in vasomotor tone in congestive heart failure

3 November 2019

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Factors contributing to the exercise limitation of heart failure

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Endothelium-dependent vasodilation is attenuated in patients with heart failure

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Effects of L-arginine on impaired acetylcholine-induced and ischemic vasodilation of the forearm in patients with heart failure.

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Impaired endothelium-mediated vasodilation in the peripheral vasculature of patients with congestive heart failure

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Basic mechanisms in congestive heart failure. Recognizing the role of proinflammatory cytokines

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Circulating levels of cytokines and their endogenous modulators in patients with mild to severe congestive heart failure due to coronary artery disease or hypertension

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Elevated circulating levels of tumor necrosis factor in severe chronic heart failure

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Tumor Necrosis Factor Soluble Receptors in Patients With Various Degrees of Congestive Heart Failure

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Elevated circulating levels of C-C chemokines in patients with congestive heart failure

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Suppression of apoptosis by nitric oxide via inhibition of interleukin-1beta-converting enzyme (ICE)-like and cysteine protease protein (CPP)-32-like proteases

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Shear stress inhibits H2O2-induced apoptosis of human endothelial cells by modulation of the glutathione redox cycle and nitric oxide synthase

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Lipopolysaccharide induces disseminated endothelial apoptosis requiring ceramide generation

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD

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Serum from patients with severe heart failure downregulates eNOS and is proapoptotic: role of tumor necrosis factor-alpha

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7 January 2021

The effect of carvedilol on morbidity and mortality in patients with chronic heart failure. U.S. Carvedilol Heart Failure Study Group

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Fluid shear stress stimulates phosphorylation of Akt in human endothelial cells: involvement in suppression of apoptosis

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The significance of shed membrane particles during programmed cell death in vitro, and in vivo, in HIV-1 infection

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Cyclosporin A inhibits apoptosis of human endothelial cells by preventing release of cytochrome C from mitochondria.

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Possible involvement of stress-activated protein kinase signaling pathway and Fas receptor expression in prevention of ischemia/reperfusion-induced cardiomyocyte apoptosis by carvedilol

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7 January 2021

Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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Tumor necrosis factor-alpha and cardiomyopathy

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Oxidized low-density lipoprotein induces apoptosis of human endothelial cells by activation of CPP32-like proteases. A mechanistic clue to the 'response to injury' hypothesis.

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Prospective, randomized comparison of effect of long-term treatment with metoprolol or carvedilol on symptoms, exercise, ejection fraction, and oxidative stress in heart failure

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Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11127444%20AND%20SRC:MED&resulttype=core&format=json

3 November 2019

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