Amino acid mutations of the infectious clone from Chinese EIAV attenuated vaccine resulted in reversion of virulence (Q58361924)

1

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Hua Liang

2

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Xiao Tong

3

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Xiujuan Fan

4

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Yan Ma

6

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Wenhua Xiang

7

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Rongxian Shen

8

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Xiaoyan Zhang

9

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Yiming Shao

Human T-cell lymphotropic virus type III shares sequence homology with a family of pathogenic lentiviruses

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language of work or name

English

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publication date

February 2006

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number of pages

12

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based on heuristic

inferred from page(s)

published in

Vaccine

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volume

24

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page(s)

738-749

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issue

6

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cites work

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Equine Infectious Anemia Virus (EIAV): what has HIV's country cousin got to tell us?

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Tumor necrosis factor-alpha production and disease severity after immunization with enriched major core protein (p26) and/or infection with equine infectious anemia virus

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SU proteins from virulent and avirulent EIAV demonstrate distinct biological properties

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Influence of long terminal repeat and env on the virulence phenotype of equine infectious anemia virus

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Antiviral, Anti-glycoprotein and Neutralizing Antibodies in Foals with Equine Infectious Anaemia Virus

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Presentation and binding affinity of equine infectious anemia virus CTL envelope and matrix protein epitopes by an expressed equine classical MHC class I molecule

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Equine infectious anemia virus genomic evolution in progressor and nonprogressor ponies.

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Equine monocyte-derived macrophage cultures and their applications for infectivity and neutralization studies of equine infectious anemia virus

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Immune responses and viral replication in long-term inapparent carrier ponies inoculated with equine infectious anemia virus

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Enhancement of EIAV replication and disease by immunization with a baculovirus-expressed recombinant envelope surface glycoprotein

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Site-directed mutagenesis by overlap extension using the polymerase chain reaction

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Assembly and release of HIV-1 precursor Pr55gag virus-like particles from recombinant baculovirus-infected insect cells

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HIV-1 infection of non-dividing cells

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A nuclear localization signal within HIV-1 matrix protein that governs infection of non-dividing cells

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Myristoylation-dependent replication and assembly of human immunodeficiency virus 1

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Cryo-electron microscopy reveals ordered domains in the immature HIV-1 particle

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Organization of immature human immunodeficiency virus type 1

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Efficient HIV-1 replication can occur in the absence of the viral matrix protein

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Structure of equine infectious anemia virus matrix protein

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Functional roles of equine infectious anemia virus Gag p9 in viral budding and infection

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Resistance to nucleoside analog reverse transcriptase inhibitors mediated by human immunodeficiency virus type 1 p6 protein

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Intracellular transport and virion incorporation of vpx requires interaction with other virus type-specific components.

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Nuclear import and cell cycle arrest functions of the HIV-1 Vpr protein are encoded by two separate genes in HIV-2/SIV(SM).

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The S2 gene of equine infectious anemia virus is a highly conserved determinant of viral replication and virulence properties in experimentally infected ponies

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Mutations occurring during serial passage of Japanese equine infectious anemia virus in primary horse macrophages

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Live attenuated, nef-deleted SIV is pathogenic in most adult macaques after prolonged observation

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Incorporation of Vpr into human immunodeficiency virus type 1 virions: requirement for the p6 region of gag and mutational analysis.

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inferred from DOI database lookup

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based on heuristic

Genomic quasispecies associated with the initiation of infection and disease in ponies experimentally infected with equine infectious anemia virus

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