The post-prandial state in Type 2 diabetes and endothelial dysfunction: effects of insulin aspart (Q58829172)

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24 November 2019

title

The post-prandial state in Type 2 diabetes and endothelial dysfunction: effects of insulin aspart (English)

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24 November 2019

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1

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author name string

A Cavarape

2

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24 November 2019

L Martinelli

3

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R Da Ros

4

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24 November 2019

G Marra

5

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L Quagliaro

6

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L Piconi

7

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R Assaloni

8

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E Motz

9

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publication date

1 February 2004

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24 November 2019

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volume

21

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issue

2

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page(s)

171-175

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Morbidity and mortality in diabetics in the Framingham population. Sixteen year follow-up study.

24 November 2019

cites work

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The pathogenesis of atherosclerosis: a perspective for the 1990s

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Hyperglycemia counterbalances the antihypertensive effect of glutathione in diabetic patients: evidence linking hypertension and glycemia through the oxidative stress in diabetes mellitus.

21 January 2018

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Vascular effects of acute hyperglycemia in humans are reversed by L-arginine. Evidence for reduced availability of nitric oxide during hyperglycemia

21 January 2018

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Hyperglycemia rapidly suppresses flow-mediated endothelium-dependent vasodilation of brachial artery.

21 January 2018

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Ascorbate restores endothelium-dependent vasodilation impaired by acute hyperglycemia in humans

21 January 2018

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Atherogenesis: a postprandial phenomenon

21 January 2018

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The emerging role of post-prandial hyperglycaemic spikes in the pathogenesis of diabetic complications

21 January 2018

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Insulin analogs with improved pharmacokinetic profiles

21 January 2018

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Postprandial glycemic control with biphasic insulin aspart in patients with type 1 diabetes

21 January 2018

1 reference

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Evidence for an independent and cumulative effect of postprandial hypertriglyceridemia and hyperglycemia on endothelial dysfunction and oxidative stress generation: effects of short- and long-term simvastatin treatment

21 January 2018

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Prognostic significance of endothelial dysfunction in hypertensive patients

21 January 2018

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10.1111/J.1464-5491.2004.01101.X

21 January 2018

Identifiers

DOI

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24 November 2019

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