NO-induced neuroprotection in ischemic preconditioning stimulates mitochondrial Mn-SOD activity and expression via RAS/ERK1/2 pathway (Q59265517)

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3 January 2020

title

NO-induced neuroprotection in ischemic preconditioning stimulates mitochondrial Mn-SOD activity and expression via Ras/ERK1/2 pathway (English)

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3

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Carmela Dell'Aversano

4

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Lucio Annunziato

9

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author name string

A Scorziello

1

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M Santillo

2

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R Sirabella

5

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S Damiano

6

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L M T Canzoniero

7

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G F Di Renzo

8

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language of work or name

English

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publication date

6 August 2007

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Journal of Neurochemistry

3 January 2020

published in

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volume

103

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issue

4

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page(s)

1472-1480

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In the neuronal cell line SH-SY5Y, oxidative stress-induced free radical overproduction causes cell death without any participation of intracellular Ca(2+) increase.

3 January 2020

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The protective effect of hypoxic preconditioning on cortical neuronal cultures is associated with increases in the activity of several antioxidant enzymes

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Provoking neuroprotection by peroxynitrite.

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Ischemic preconditioning preserves mitochondrial function after global cerebral ischemia in rat hippocampus

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Generation of hydrogen peroxide during brief oxygen-glucose deprivation induces preconditioning neuronal protection in primary cultured neurons

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Requirement for nitric oxide activation of p21(ras)/extracellular regulated kinase in neuronal ischemic preconditioning

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Analysis of nitrate, nitrite, and [15N]nitrate in biological fluids

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Re-examination and further development of a precise and rapid dye method for measuring cell growth/cell kill

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Mitochondrial signal lacking manganese superoxide dismutase failed to prevent cell death by reoxygenation following hypoxia in a human pancreatic cancer cell line, KP4.

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Hypoxic preconditioning induces changes in HIF-1 target genes in neonatal rat brain

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Immunohistochemical localization of superoxide dismutase in the hippocampus following ischemia in a gerbil model of ischemic tolerance.

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'Ischemic tolerance' phenomenon found in the brain

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‘Ischemic tolerance’ phenomenon detected in various brain regions

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Epsilon protein kinase C mediated ischemic tolerance requires activation of the extracellular regulated kinase pathway in the organotypic hippocampal slice

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Activation of mitochondrial ATP-dependent potassium channels protects neurons against ischemia-induced death by a mechanism involving suppression of Bax translocation and cytochrome c release

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Mechanisms of cardiac preconditioning: ten years after the discovery of ischemic preconditioning

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CREB-mediated Bcl-2 protein expression after ischemic preconditioning.

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Rapid degradation of Bim by the ubiquitin-proteasome pathway mediates short-term ischemic tolerance in cultured neurons

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Manganese Superoxide Dismutase Affects Cytochrome c Release and Caspase-9 Activation After Transient Focal Cerebral Ischemia in Mice

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The type and the localization of cAMP-dependent protein kinase regulate transmission of cAMP signals to the nucleus in cortical and cerebellar granule cells

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Identifiers

DOI

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3 January 2020

1 reference