Mechanism of nasal tolerance induced by a recombinant fragment of acetylcholine receptor for treatment of experimental myasthenia gravis (Q59302473)

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1 November 2019

title

Mechanism of nasal tolerance induced by a recombinant fragment of acetylcholine receptor for treatment of experimental myasthenia gravis (English)

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1 November 2019

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1

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1 November 2019

author name string

D Barchan

series ordinal

2

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1 November 2019

S Fuchs

series ordinal

3

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1 November 2019

M C Souroujon

series ordinal

4

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1 November 2019

publication date

1 November 2000

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Journal of Neuroimmunology

1 November 2019

published in

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1 November 2019

volume

111

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1 November 2019

issue

1-2

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1 November 2019

page(s)

161-168

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Immunochemical studies on acetylcholine receptor from Torpedo californica

1 November 2019

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Antigen-driven peripheral immune tolerance: suppression of experimental autoimmmune encephalomyelitis and collagen-induced arthritis by aerosol administration of myelin basic protein or type II collagen.

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Interferon gamma (IFN-gamma) is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice

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Antigen-specific modulation of experimental myasthenia gravis: nasal tolerization with recombinant fragments of the human acetylcholine receptor alpha-subunit

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Differential activities of immunogenic collagen type II peptides in the induction of nasal tolerance to collagen-induced arthritis

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Nasal administration of retinal antigens suppresses the inflammatory response in experimental allergic uveoretinitis. A preliminary report of intranasal induction of tolerance with retinal antigens

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The CD40 ligand. At the center of the immune universe?

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Suppression of ongoing experimental myasthenia by oral treatment with an acetylcholine receptor recombinant fragment.

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Prevention of experimental myasthenia gravis by nasal administration of synthetic acetylcholine receptor T epitope sequences

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B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: application to autoimmune disease therapy

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Nasal tolerance to experimental autoimmune myasthenia gravis: tolerance reversal by nasal administration of minute amounts of interferon-gamma

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Cellular mRNA expression of interferon-gamma (IFN-gamma), IL-4 and transforming growth factor-beta (TGF-beta) in rats nasally tolerized against experimental autoimmune myasthenia gravis (EAMG)

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Suppression of experimental autoimmune myasthenia gravis by nasal administration of acetylcholine receptor

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Intranasal administration of denatured type II collagen and its fragments can delay the onset of collagen-induced arthritis

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The natural immune response to inhaled soluble protein antigens involves major histocompatibility complex (MHC) class I-restricted CD8+ T cell-mediated but MHC class II-restricted CD4+ T cell-dependent immune deviation resulting in selective suppres

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IL-12 is involved in the induction of experimental autoimmune myasthenia gravis, an antibody-mediated disease

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7 January 2021

Suppression of murine collagen-induced arthritis by nasal administration of collagen

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7 January 2021

Oral administration of acetylcholine receptor: effects on experimental myasthenia gravis

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7 January 2021

Nasal tolerance in experimental autoimmune myasthenia gravis (EAMG): induction of protective tolerance in primed animals

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7 January 2021

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7 January 2021

Suppression of experimental autoimmune myasthenia gravis by oral administration of acetylcholine receptor

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Identifiers

DOI

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1 November 2019

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