Two Naturally Occurring Insulin Receptor Tyrosine Kinase Domain Mutants Provide Evidence That Phosphoinositide 3-Kinase Activation Alone Is Not Sufficient for the Mediation of Insulin’s Metabolic and Mitogenic Effects (Q60677537)

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scientific article published in Journal of Biological Chemistry
  • Two naturally occurring insulin receptor tyrosine kinase domain mutants provide evidence that phosphoinositide 3-kinase activation alone is not sufficient for the mediation of insulin's metabolic and mitogenic effects
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English
Two Naturally Occurring Insulin Receptor Tyrosine Kinase Domain Mutants Provide Evidence That Phosphoinositide 3-Kinase Activation Alone Is Not Sufficient for the Mediation of Insulin’s Metabolic and Mitogenic Effects
scientific article published in Journal of Biological Chemistry
  • Two naturally occurring insulin receptor tyrosine kinase domain mutants provide evidence that phosphoinositide 3-kinase activation alone is not sufficient for the mediation of insulin's metabolic and mitogenic effects

Statements

Two Naturally Occurring Insulin Receptor Tyrosine Kinase Domain Mutants Provide Evidence That Phosphoinositide 3-Kinase Activation Alone Is Not Sufficient for the Mediation of Insulin’s Metabolic and Mitogenic Effects (English)
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Two naturally occurring insulin receptor tyrosine kinase domain mutants provide evidence that phosphoinositide 3-kinase activation alone is not sufficient for the mediation of insulin's metabolic and mitogenic effects (English)
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Anna Krook
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Jonathan P. Whitehead
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Stephen P. Dobson
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Matthew R. Griffiths
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Margrite Ouwens
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Claire Baker
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Amanda C. Hayward
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Sankhya K. Sen
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J. Antoine Maassen
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Kenneth Siddle
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Jeremy M. Tavaré
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Stephen O’Rahilly
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A Krook
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J P Whitehead
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S P Dobson
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M R Griffiths
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M Ouwens
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C Baker
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A C Hayward
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S K Sen
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J A Maassen
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K Siddle
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J M Tavaré
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S O'Rahilly
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28 November 1997
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272
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48
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30208-30214
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30208-14
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