SG formation relies on eIF4GI-G3BP interaction which is targeted by picornavirus stress antagonists (Q60922523)

1

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Zhulong Hu

2

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Qiang Zhang

3

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Shanshan Fan

4

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Yi Zhong

5

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Dong Guo

6

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Yali Qin

7

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Mingzhou Chen

Creative Commons Attribution 4.0 International

8

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language of work or name

English

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publication date

2019

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published in

Cell discovery

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volume

5

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issue

1

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page(s)

1

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copyright license

start time

1 January 2019

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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exact match

https://scigraph.springernature.com/pub.10.1038/s41421-018-0068-4

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cites work

Stress granules: the Tao of RNA triage

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Dynamic shuttling of TIA-1 accompanies the recruitment of mRNA to mammalian stress granules

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

The RasGAP-associated endoribonuclease G3BP assembles stress granules

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Stress granule assembly is mediated by prion-like aggregation of TIA-1

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Nuclear relocalisation of cytoplasmic poly(A)-binding proteins PABP1 and PABP4 in response to UV irradiation reveals mRNA-dependent export of metazoan PABPs

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Diversion of stress granules and P-bodies during viral infection

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Cytoplasmic RNA Granules and Viral Infection

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Sequestration of G3BP coupled with efficient translation inhibits stress granules in Semliki Forest virus infection

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Chikungunya virus nsP3 blocks stress granule assembly by recruitment of G3BP into cytoplasmic foci

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Interaction of TIA-1/TIAR with West Nile and dengue virus products in infected cells interferes with stress granule formation and processing body assembly

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Novel Staufen1 ribonucleoproteins prevent formation of stress granules but favour encapsidation of HIV-1 genomic RNA.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Japanese encephalitis virus core protein inhibits stress granule formation through an interaction with Caprin-1 and facilitates viral propagation

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Inclusion bodies of human parainfluenza virus type 3 inhibit antiviral stress granule formation by shielding viral RNAs.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Human enterovirus 71 and hand, foot and mouth disease.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Inhibition of cytoplasmic mRNA stress granule formation by a viral proteinase

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Production of a dominant-negative fragment due to G3BP1 cleavage contributes to the disruption of mitochondria-associated protective stress granules during CVB3 infection

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Encephalomyocarditis virus disrupts stress granules, the critical platform for triggering antiviral innate immune responses

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

The leader protein of cardioviruses inhibits stress granule assembly.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

MDA5 localizes to stress granules, but this localization is not required for the induction of type I interferon

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Picornavirus 2A protease regulates stress granule formation to facilitate viral translation.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Enhanced enteroviral infectivity via viral protease-mediated cleavage of Grb2-associated binder 1.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Additive Promotion of Viral Internal Ribosome Entry Site-Mediated Translation by Far Upstream Element-Binding Protein 1 and an Enterovirus 71-Induced Cleavage Product.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

A single amino acid change in protein synthesis initiation factor 4G renders cap-dependent translation resistant to picornaviral 2A proteases

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

A thermosensitive mutant of HRV2 2A proteinase: evidence for direct cleavage of eIF4GI and eIF4GII

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Selective Removal of FG Repeat Domains from the Nuclear Pore Complex by Enterovirus 2A(pro)

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Specific cleavage of the nuclear pore complex protein Nup62 by a viral protease

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Modulation of enteroviral proteinase cleavage of poly(A)-binding protein (PABP) by conformation and PABP-associated factors

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Dominant-negative function of the C-terminal fragments of NBR1 and SQSTM1 generated during enteroviral infection

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Cleavage of serum response factor mediated by enteroviral protease 2A contributes to impaired cardiac function

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Both G3BP1 and G3BP2 contribute to stress granule formation

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Mass spectrometric analysis of the N terminus of translational initiation factor eIF4G-1 reveals novel isoforms

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

A tale of two termini: a functional interaction between the termini of an mRNA is a prerequisite for efficient translation initiation

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

The mRNA 5' cap-binding protein eIF4E and control of cell growth

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Eukaryotic translation initiation factor 4E (eIF4E) binding site and the middle one-third of eIF4GI constitute the core domain for cap-dependent translation, and the C-terminal one-third functions as a modulatory region

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

A novel functional human eukaryotic translation initiation factor 4G

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Proteolysis of human eukaryotic translation initiation factor eIF4GII, but not eIF4GI, coincides with the shutoff of host protein synthesis after poliovirus infection

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

G3BP-Caprin1-USP10 complexes mediate stress granule condensation and associate with 40S subunits

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Casein Kinase 2 Is Linked to Stress Granule Dynamics through Phosphorylation of the Stress Granule Nucleating Protein G3BP1.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Arginine Demethylation of G3BP1 Promotes Stress Granule Assembly

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Viral and cellular proteins containing FGDF motifs bind G3BP to block stress granule formation

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Principles and Properties of Stress Granules

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Distinct stages in stress granule assembly and disassembly

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

ATPase-Modulated Stress Granules Contain a Diverse Proteome and Substructure.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Hyperthermia stress activates heat shock protein expression via propyl isomerase 1 regulation with heat shock factor 1.

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Coordination of translational control and protein homeostasis during severe heat stress

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Improved vectors and genome-wide libraries for CRISPR screening

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Generation of neutralizing monoclonal antibodies against Enterovirus 71 using synthetic peptides

1 reference

https://api.crossref.org/works/10.1038%2FS41421-018-0068-4

7 January 2021

Mapping the cleavage site in protein synthesis initiation factor eIF-4 gamma of the 2A proteases from human Coxsackievirus and rhinovirus

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/30603102

12 December 2020

inferred from PubMed ID database lookup

Class I and II Small Heat Shock Proteins Together with HSP101 Protect Protein Translation Factors during Heat Stress

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/30603102

12 December 2020