Ability of T cells from patients with rheumatoid arthritis to respond to immunoglobulin G (Q61912615)

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L M Macht

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D C Wraith

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C J Elson

The carrier effect in the secondary response to hapten-protein conjugates. V. Use of antilymphocyte serum to deplete animals of helper cells

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language of work or name

English

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publication date

September 1999

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published in

Immunology

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volume

98

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issue

1

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page(s)

116-22

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cites work

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Primary and secondary human in vitro T-cell responses to soluble antigens are mediated by subsets bearing different CD45 isoforms

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Auto-antigens in rheumatoid arthritis--a new candidate?

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“Homozygosity” for the HLA–DR shared epitope contributes the highest risk for rheumatoid arthritis concordance in identical twins

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Generation and molecular analyses of two rheumatoid synovial fluid-derived IgG rheumatoid factors.

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Autoimmunity to type II collagen an experimental model of arthritis

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B- and T-cell autoantigens in pristane-induced arthritis

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Expression of mammalian 60-kD heat shock protein in the joints of mice with pristane-induced arthritis

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Inhibition of experimental autoimmune encephalomyelitis by inhalation but not oral administration of the encephalitogenic peptide: influence of MHC binding affinity

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Multiple self epitopes on the Rhesus polypeptides stimulate immunologically ignorant human T cells in vitro

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Gamma globulin complexes in synovial fluids of patients with rheumatoid arthritis. Partial characterization and relationship to lowered complement levels

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Immunological help at last.

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Divergent T-cell cytokine patterns in inflammatory arthritis

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T cell responses to human type II collagen in patients with rheumatoid arthritis and healthy controls

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Joint-derived T cells in rheumatoid arthritis react with self-immunoglobulin heavy chains or immunoglobulin-binding proteins that copurify with immunoglobulin

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An immunohistological analysis of lymphocyte subpopulations and their microenvironment in the synovial membranes of patients with rheumatoid arthritis using monoclonal antibodies

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Peptide-induced nasal tolerance for a mycobacterial heat shock protein 60 T cell epitope in rats suppresses both adjuvant arthritis and nonmicrobially induced experimental arthritis

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Quantitative analysis of cytokine gene expression in rheumatoid arthritis

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Determination of lymphocyte division by flow cytometry

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Epitope specificity and MHC restriction of rheumatoid arthritis synovial T cell clones which recognize a mycobacterial 65 kDa heat shock protein.

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Persistence of collagen type II-specific T-cell clones in the synovial membrane of a patient with rheumatoid arthritis

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Mucosal tolerance and suppression of collagen-induced arthritis (CIA) induced by nasal inhalation of synthetic peptide 184-198 of bovine type II collagen (CII) expressing a dominant T cell epitope

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Immunologically ignorant autoreactive T cells, epitope spreading and repertoire limitation

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Rheumatoid factor plaque-forming cells in rheumatoid synovial tissue

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Complement-Fixing Intracellular Complexes of IgG Rheumatoid Factor in Rheumatoid Plasma Cells

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Detection of cytokines at the site of tuberculin-induced delayed-type hypersensitivity in man.

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Peptide recognition, T cell receptor usage and HLA restriction elements of human heat-shock protein (hsp) 60 and mycobacterial 65-kDa hsp-reactive T cell clones from rheumatoid synovial fluid

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An immunodominant epitope from mycobacterial 65-kDa heat shock protein protects against pristane-induced arthritis

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Activation of T Cells by Autoantigen Immobilized by Specific Antibodies

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Human cartilage glycoprotein-39 as a candidate autoantigen in rheumatoid arthritis

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New roles for rheumatoid factor

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Antigen-induced arthritis in mice. I. Induction of arthritis in various strains of mice.

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The development of monoclonal antibodies to the human mitochondrial 60-kd heat-shock protein, and their use in studying the expression of the protein in rheumatoid arthritis

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Expectation bias in rheumatoid arthritis clinical trials. The anti-CD4 monoclonal antibody experience

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Cytokine expression in rheumatoid arthritis

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The shared epitope hypothesis. An approach to understanding the molecular genetics of susceptibility to rheumatoid arthritis

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Stimulation of lymphocyte transformation by autologous serum fractions from patients with rheumatoid arthritis.

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