Implications of maturation for viral gene delivery to skeletal muscle (Q62971278)

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S.Steven Floyd

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David K Booth

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Thomas Oligino

4

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David Krisky

5

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Peggy Marconi

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Joseph C Glorioso

7

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Johnny Huard

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J C van Deutekom

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S S Floyd

2

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D K Booth

3

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T Oligino

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D Krisky

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P Marconi

6

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J C Glorioso

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J Huard

Systemic delivery of recombinant proteins by genetically modified myoblasts

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language of work or name

English

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publication date

May 1998

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published in

Neuromuscular Disorders

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volume

8

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issue

3-4

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page(s)

135-148

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135-48

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Results of a triple blind clinical study of myoblast transplantations without immunosuppressive treatment in young boys with Duchenne muscular dystrophy.

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Human dystrophin expression in mdx mice after intramuscular injection of DNA constructs

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Dystrophin expression in muscles of mdx mice after adenovirus-mediated in vivo gene transfer

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The basal lamina is a physical barrier to herpes simplex virus-mediated gene delivery to mature muscle fibers.

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Impairment of force generation after adenovirus-mediated gene transfer to muscle is alleviated by adenoviral gene inactivation and host CD8+ T cell deficiency

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Viral gene delivery to skeletal muscle: insights on maturation-dependent loss of fiber infectivity for adenovirus and herpes simplex type 1 viral vectors

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Plasmid DNA is superior to viral vectors for direct gene transfer into adult mouse skeletal muscle

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Ex vivo gene transfer using adenovirus-mediated full-length dystrophin delivery to dystrophic muscles

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Immunosuppression by FK506 markedly prolongs expression of adenovirus-delivered transgene in skeletal muscles of adult dystrophic [mdx] mice

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FK506 immunosuppression to control the immune reactions triggered by first-generation adenovirus-mediated gene transfer

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Recombinant IL-12 prevents formation of blocking IgA antibodies to recombinant adenovirus and allows repeated gene therapy to mouse lung.

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Assembly of basement membranes

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Recombinant adeno-associated viral vectors mediate long-term transgene expression in muscle

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