Antagonism of dsRNA-Induced Innate Immune Pathways by NS4a and NS4b Accessory Proteins during MERS Coronavirus Infection (Q64120102)

6

object named as

Susan R Weiss

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Stephen A Goldstein

2

object named as

Stephen A Goldstein

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Boyd L Yount

4

object named as

Boyd Yount

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author name string

Courtney E Comar

1

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Yize Li

In vitro cultivation of human tumors: establishment of cell lines derived from a series of solid tumors

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language of work or name

English

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publication date

26 March 2019

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published in

mBio

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volume

10

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issue

2

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cites work

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Middle East respiratory syndrome coronavirus accessory protein 4a is a type I interferon antagonist

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Reverse genetics with a full-length infectious cDNA of the Middle East respiratory syndrome coronavirus

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The structural and accessory proteins M, ORF 4a, ORF 4b, and ORF 5 of Middle East respiratory syndrome coronavirus (MERS-CoV) are potent interferon antagonists

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The ORF4b-encoded accessory proteins of Middle East respiratory syndrome coronavirus and two related bat coronaviruses localize to the nucleus and inhibit innate immune signalling

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Middle east respiratory syndrome coronavirus 4a protein is a double-stranded RNA-binding protein that suppresses PACT-induced activation of RIG-I and MDA5 in the innate antiviral response.

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Murine AKAP7 has a 2',5'-phosphodiesterase domain that can complement an inactive murine coronavirus ns2 gene.

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MERS-CoV in Upper Respiratory Tract and Lungs of Dromedary Camels, Saudi Arabia, 2013-2014

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Middle East Respiratory Syndrome Coronavirus nsp1 Inhibits Host Gene Expression by Selectively Targeting mRNAs Transcribed in the Nucleus while Sparing mRNAs of Cytoplasmic Origin

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Middle East respiratory syndrome coronavirus ORF4b protein inhibits type I interferon production through both cytoplasmic and nuclear targets

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A phylogenetically distinct Middle East respiratory syndrome coronavirus detected in a dromedary calf from a closed dairy herd in Dubai with rising seroprevalence with age.

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Activation of RNase L is dependent on OAS3 expression during infection with diverse human viruses

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Deletion Variants of Middle East Respiratory Syndrome Coronavirus from Humans, Jordan, 2015.

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Middle East Respiratory Syndrome Coronavirus NS4b Protein Inhibits Host RNase L Activation.

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Middle East Respiratory Coronavirus Accessory Protein 4a Inhibits PKR-Mediated Antiviral Stress Responses

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Lineage A Betacoronavirus NS2 Proteins and the Homologous Torovirus Berne pp1a Carboxy-Terminal Domain Are Phosphodiesterases That Antagonize Activation of RNase L.

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Early endonuclease-mediated evasion of RNA sensing ensures efficient coronavirus replication

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Further Evidence for Bats as the Evolutionary Source of Middle East Respiratory Syndrome Coronavirus

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Coronavirus nonstructural protein 15 mediates evasion of dsRNA sensors and limits apoptosis in macrophages

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MERS-CoV Accessory ORFs Play Key Role for Infection and Pathogenesis

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MERS-CoV 4b protein interferes with the NF-κB-dependent innate immune response during infection.

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MERS coronaviruses from camels in Africa exhibit region-dependent genetic diversity.

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A metagenomic viral discovery approach identifies potential zoonotic and novel mammalian viruses in Neoromicia bats within South Africa.

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Inhibition of Stress Granule Formation by Middle East Respiratory Syndrome Coronavirus 4a Accessory Protein Facilitates Viral Translation, Leading to Efficient Virus Replication

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Structural and mechanistic basis for preferential deadenylation of U6 snRNA by Usb1.

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Type III Interferons in Antiviral Defenses at Barrier Surfaces

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