HB-EGF induces COL7A1 expression in keratinocytes and fibroblasts: possible mechanism underlying allogeneic fibroblast therapy in recessive dystrophic epidermolysis Bullosa. (Q64939511)

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scientific article published on 7 April 2011

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English	HB-EGF induces COL7A1 expression in keratinocytes and fibroblasts: possible mechanism underlying allogeneic fibroblast therapy in recessive dystrophic epidermolysis Bullosa.	scientific article published on 7 April 2011

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27 January 2020

HB-EGF induces COL7A1 expression in keratinocytes and fibroblasts: possible mechanism underlying allogeneic fibroblast therapy in recessive dystrophic epidermolysis Bullosa (English)

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27 January 2020

recessive dystrophic epidermolysis bullosa

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inferred from title

2

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27 January 2020

7

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27 January 2020

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Nikoletta Nagy

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27 January 2020

Akio Tanaka

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27 January 2020

Joey E Lai-Cheong

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27 January 2020

Tanasit Techanukul

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27 January 2020

Jemima E Mellerio

6

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27 January 2020

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7 April 2011

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27 January 2020

Journal of Investigative Dermatology

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27 January 2020

131

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27 January 2020

8

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27 January 2020

1771-1774

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27 January 2020

https://scigraph.springernature.com/pub.10.1038/jid.2011.85

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Opposite Smad and chicken ovalbumin upstream promoter transcription factor inputs in the regulation of the collagen VII gene promoter by transforming growth factor-beta

1 reference

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Replenishment of type VII collagen and re-epithelialization of chronically ulcerated skin after intradermal administration of allogeneic mesenchymal stromal cells in two patients with recessive dystrophic epidermolysis bullosa

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The classification of inherited epidermolysis bullosa (EB): Report of the Third International Consensus Meeting on Diagnosis and Classification of EB

1 reference

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A hypomorphic mouse model of dystrophic epidermolysis bullosa reveals mechanisms of disease and response to fibroblast therapy

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Phorbol ester induces the rapid processing of cell surface heparin-binding EGF-like growth factor: conversion from juxtacrine to paracrine growth factor activity

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Heparin-binding EGF-like growth factor: a juxtacrine growth factor

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Mechanisms of fibroblast cell therapy for dystrophic epidermolysis bullosa: high stability of collagen VII favors long-term skin integrity

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Matrix metalloproteinase-7 activates heparin-binding epidermal growth factor-like growth factor in cutaneous squamous cell carcinoma.

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UVA-340 as energy source, mimicking natural sunlight, activates the transcription factor AP-1 in cultured fibroblasts: evidence for involvement of protein kinase-C.

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Injection of genetically engineered fibroblasts corrects regenerated human epidermolysis bullosa skin tissue

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Epidermolysis bullosa acquisita antigen is synthesized by both human keratinocytes and human dermal fibroblasts

1 reference

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Potential of fibroblast cell therapy for recessive dystrophic epidermolysis bullosa.

1 reference

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Normal and gene-corrected dystrophic epidermolysis bullosa fibroblasts alone can produce type VII collagen at the basement membrane zone

1 reference

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Fibroblast-based cell therapy strategy for recessive dystrophic epidermolysis bullosa

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