Steroids decrease granulocyte membrane fluidity, while phorbol ester increases membrane fluidity. Studies using electron paramagnetic resonance (Q68455170)

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scientific article published on 01 February 1990

Language	Label	Description	Also known as
English	Steroids decrease granulocyte membrane fluidity, while phorbol ester increases membrane fluidity. Studies using electron paramagnetic resonance	scientific article published on 01 February 1990

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scholarly article

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24 September 2019

Steroids decrease granulocyte membrane fluidity, while phorbol ester increases membrane fluidity. Studies using electron paramagnetic resonance (English)

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24 September 2019

author name string

H R Lamche

1

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P T Silberstein

2

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A C Knabe

3

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D D Thomas

4

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H S Jacob

5

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D E Hammerschmidt

6

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publication date

1 February 1990

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1 reference

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14

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1

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61-70

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https://scigraph.springernature.com/pub.10.1007/bf00914030

0 references

Inhibition of arachidonic acid release from cells as the biochemical action of anti-inflammatory corticosteroids

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

12 December 2020

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Complement-induced granulocyte aggregation: an unsuspected mechanism of disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

12 December 2020

based on heuristic

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Corticosteroids inhibit complement-induced granulocyte aggregation. A possible mechanism for their efficacy in shock states

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

12 December 2020

based on heuristic

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Synergy among agents inhibiting granulocyte aggregation

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

12 December 2020

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A controlled clinical trial of high-dose methylprednisolone in the treatment of severe sepsis and septic shock

1 reference

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Serum complement levels in canine endotoxin shock: relation to survival and to corticosteroid therapy

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

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Corticosteroids reversibly inhibit chemotactic peptide-receptor binding and granulocyte response, yet allow desensitization and receptor down-regulation

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

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Interactions of immunoglobulins with liposomes: An ESR and diffusion study demonstrating protection by hydrocortisone

1 reference

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Effect of high-dose glucocorticoid therapy on mortality in patients with clinical signs of systemic sepsis

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

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Spin-label measurements in membranes. With appendix: a use of computers in EPR spectroscopy.

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

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Membrane Fluidity in Human and Mouse Chediak-Higashi Leukocytes

1 reference

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Corticosteroids block binding of chemotactic peptide to its receptor on granulocytes and cause disaggregation of granulocyte aggregates in vitro

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

12 December 2020

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Fat embolism prophylaxis with corticosteroids. A prospective study in high-risk patients

1 reference

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12 December 2020

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Inhibition of granulocyte function by steroids is not limited to corticoids. Studies with sex steroids

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

12 December 2020

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Steroids in the treatment of clinical septic shock

1 reference

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Effects of ibuprofen on chemotactic peptide-receptor binding and granulocyte response

1 reference

https://pubmed.ncbi.nlm.nih.gov/2157660

12 December 2020

based on heuristic

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Methylprednisolone. Pharmacologic doses in shock lung syndrome

1 reference

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A rapid method for purification of human granulocytes using percoll. A comparison with dextran sedimentation.

1 reference

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Macrocortin: a polypeptide causing the anti-phospholipase effect of glucocorticoids

1 reference

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12 December 2020

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Identifiers

10.1007/BF00914030

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:2157660%20AND%20SRC:MED&resulttype=core&format=json

24 September 2019

1 reference

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24 September 2019

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