Recovery phase of acute experimental autoimmune encephalomyelitis in rats corresponds to development of endothelial cell unresponsiveness to interferon gamma activation (Q71302847)

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scientific article published on 01 May 1996

Language	Label	Description	Also known as
English	Recovery phase of acute experimental autoimmune encephalomyelitis in rats corresponds to development of endothelial cell unresponsiveness to interferon gamma activation	scientific article published on 01 May 1996

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scholarly article

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17 October 2019

Recovery phase of acute experimental autoimmune encephalomyelitis in rats corresponds to development of endothelial cell unresponsiveness to interferon gamma activation (English)

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17 October 2019

experimental autoimmune encephalomyelitis

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author name string

P Dore-Duffy

1

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17 October 2019

R Balabanov

2

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17 October 2019

J Rafols

3

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17 October 2019

R H Swanborg

4

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17 October 2019

publication date

1 May 1996

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17 October 2019

Journal of Neuroscience Research

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17 October 2019

44

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17 October 2019

3

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17 October 2019

223-234

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17 October 2019

Primary culture of capillary endothelium from rat brain

1 reference

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Distinct cellular functions mediated by different VLA integrin alpha subunit cytoplasmic domains

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Induction and detection of a human endothelial activation antigen in vivo

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Ia antigens in the normal rat nervous system and in lesions of experimental allergic encephalomyelitis

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Microvascular pericytes contain muscle and nonmuscle actins

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Expression of Ia molecules by astrocytes during acute experimental allergic encephalomyelitis in the Lewis rat

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Susceptibility of Lewis rats to experimental autoimmune encephalomyelitis after recovery from passively induced disease.

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Experimental allergic encephalomyelitis: neutralizing antibody to TGF beta 1 enhances the clinical severity of the disease.

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The transforming growth factor-beta family

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In situ detection of class I and II major histocompatibility complex antigens in the rat central nervous system during experimental allergic encephalomyelitis. An immunohistochemical study

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Cytokine-regulated adhesion between encephalitogenic T lymphocytes and cerebrovascular endothelial cells

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Suppressor T cells generated by oral tolerization to myelin basic protein suppress both in vitro and in vivo immune responses by the release of transforming growth factor beta after antigen-specific triggering

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Evidence of endogenous regulatory function of transforming growth factor-beta 1 in experimental allergic encephalomyelitis

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Antagonistic effects of endogenous and exogenous TGF-beta and TNF on auto-immune diseases in mice

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Oral Tolerance to Myelin Basic Protein Induces Regulatory TGF-β-Secreting T Cells in Peyer's Patches of SJL Mice

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Transforming growth factors beta 1 and beta 2: cytokines with identical immunosuppressive effects and a potential role in the regulation of autoimmune T cell function

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The immunopathology of acute experimental allergic encephalomyelitis. IV. An ultrastructural immunocytochemical study of class II major histocompatibility complex molecule (Ia) expression.

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Identifiers

10.1002/(SICI)1097-4547(19960501)44:3<223::AID-JNR3>3.0.CO;2-I

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1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:8723761%20AND%20SRC:MED&resulttype=core&format=json

17 October 2019

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