Modulation of experimental autoimmune encephalomyelitis: effect of altered peptide ligand on chemokine and chemokine receptor expression (Q73056551)

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31 October 2019

title

Modulation of experimental autoimmune encephalomyelitis: effect of altered peptide ligand on chemokine and chemokine receptor expression (English)

1 reference

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experimental autoimmune encephalomyelitis

31 October 2019

main subject

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author name string

F R Fischer

1

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31 October 2019

L Santambrogio

2

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31 October 2019

Y Luo

3

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31 October 2019

M A Berman

4

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31 October 2019

W W Hancock

5

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31 October 2019

M E Dorf

6

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31 October 2019

publication date

1 October 2000

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Journal of Neuroimmunology

31 October 2019

published in

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31 October 2019

volume

110

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31 October 2019

issue

1-2

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31 October 2019

page(s)

195-208

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Chemokines and leukocyte traffic

31 October 2019

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Central nervous system chemokine mRNA accumulation follows initial leukocyte entry at the onset of acute murine experimental autoimmune encephalomyelitis

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A polymorphism in the mouse crg-2/IP-10 gene complicates chemokine gene expression analysis using a commercial ribonuclease protection assay

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MIP-1alpha and MCP-1 differentially regulate acute and relapsing autoimmune encephalomyelitis as well as Th1/Th2 lymphocyte differentiation

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CCR5 is characteristic of Th1 lymphocytes

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Chemokines--chemotactic cytokines that mediate inflammation

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Impaired B-lymphopoiesis, myelopoiesis, and derailed cerebellar neuron migration in CXCR4- and SDF-1-deficient mice

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Reduced chemokine and chemokine receptor expression in spinal cords of TCR BV8S2 transgenic mice protected against experimental autoimmune encephalomyelitis with BV8S2 protein

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Experimental autoimmune encephalomyelitis on the SJL mouse: effect of gamma delta T cell depletion on chemokine and chemokine receptor expression in the central nervous system

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The role of CCR7 in TH1 and TH2 cell localization and delivery of B cell help in vivo

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Microbial epitopes act as altered peptide ligands to prevent experimental autoimmune encephalomyelitis

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A single amino acid change in a myelin basic protein peptide confers the capacity to prevent rather than induce experimental autoimmune encephalomyelitis

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Identifiers

DOI

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31 October 2019

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