B cell-mediated infection of stimulated and unstimulated autologous T lymphocytes with HIV-1: role of complement (Q73111991)

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1 November 2019

title

B cell-mediated infection of stimulated and unstimulated autologous T lymphocytes with HIV-1: role of complement (English)

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1 November 2019

author name string

S Doepper

1

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1 November 2019

H Stoiber

2

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1 November 2019

L Kacani

3

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1 November 2019

G Sprinzl

4

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1 November 2019

F Steindl

5

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1 November 2019

W M Prodinger

6

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1 November 2019

M P Dierich

7

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1 November 2019

publication date

1 September 2000

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1 November 2019

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1 November 2019

volume

202

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1 November 2019

issue

3

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1 November 2019

page(s)

293-305

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Dendritic cells transmit human immunodeficiency virus type 1 to monocytes and monocyte-derived macrophages.

1 November 2019

cites work

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Low levels of HIV-1 infection in cutaneous dendritic cells promote extensive viral replication upon binding to memory CD4+ T cells

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HIV-1 Entry Cofactor: Functional cDNA Cloning of a Seven-Transmembrane, G Protein-Coupled Receptor

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B lymphocytes in lymph nodes and peripheral blood are important for binding immune complexes containing HIV-1

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Human immunodeficiency virus type 1 activates the classical pathway of complement by direct C1 binding through specific sites in the transmembrane glycoprotein gp41

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Binding of the B cell activation antigen B7 to CD28 costimulates T cell proliferation and interleukin 2 mRNA accumulation

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The significance of low bcl-2 expression by CD45RO T cells in normal individuals and patients with acute viral infections. The role of apoptosis in T cell memory

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Fas antigen stimulation induces marked apoptosis of T lymphocytes in human immunodeficiency virus-infected individuals.

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Evidence for a viral superantigen in humans

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T cell response to staphylococcal superantigens by asymptomatic HIV-infected individuals exhibits selective changes in T cell receptor V beta-chain usage

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HLA class II on HIV particles is functional in superantigen presentation to human T cells: implications for HIV pathogenesis

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T-cell antigen receptor V beta subsets are not preferentially deleted in AIDS.

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10.1016/S0171-2985(00)80035-2

Identifiers

DOI

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1 November 2019

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