The relationship of plasma catecholamine and lactate during anaerobic threshold exercise in mitochondrial myopathies (Q73141358)

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1 November 2019

title

The relationship of plasma catecholamine and lactate during anaerobic threshold exercise in mitochondrial myopathies (English)

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1 November 2019

1

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1 November 2019

Maria Laura Manca

3

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1 November 2019

author name string

M Renna

2

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1 November 2019

C Prontera

4

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G Zucchelli

5

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1 November 2019

E Ferrannini

6

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L Murri

7

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1 November 2019

publication date

1 October 1999

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1 November 2019

published in

Neuromuscular Disorders

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1 November 2019

volume

9

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issue

6-7

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1 November 2019

page(s)

411-416

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Mitochondrial encephalomyopathies

1 November 2019

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The expanding clinical spectrum of mitochondrial diseases

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Plasma catecholamine and lactate response during graded exercise with varied glycogen conditions

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Exercise fuel mobilization in mitochondrial myopathy: a metabolic dilemma.

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Residual muscle cytochrome c oxidase activity accounts for submaximal exercise lactate threshold in chronic progressive external ophthalmoplegia

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Influence of plasma catecholamines on the lactate threshold during graded exercise.

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Plasma catecholamines during endurance exercise of different intensities as related to the individual anaerobic threshold.

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A comparison of the blood lactate and plasma catecholamine thresholds in untrained male subjects

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Glucose administration before exercise modulates catecholaminergic responses in glycogen-depleted subjects

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Catecholamine and blood lactate responses to incremental rowing and running exercise

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Effects of adrenergic agonists and antagonists on muscle O2 uptake and lactate metabolism

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Effects of acute beta-adrenergic blockade on blood and muscle lactate concentration during submaximal exercise

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Metabolic and hormonal responses to adrenoceptor antagonists in 48-hour-starved exercising rats

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The significance of the aerobic-anaerobic transition for the determination of work load intensities during endurance training

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Sympathetic activation in exercise is not dependent on muscle acidosis. Direct evidence from studies in metabolic myopathies

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Peripheral neuropathy in mitochondrial encephalomyopathies

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Identifiers

DOI

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1 November 2019

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