Inhibition of vascular NADH/NADPH oxidase activity by thiol reagents: lack of correlation with cellular glutathione redox status (Q73153925)

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1 November 2019

title

Inhibition of vascular NADH/NADPH oxidase activity by thiol reagents: lack of correlation with cellular glutathione redox status (English)

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1 November 2019

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M Janiszewski

1

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1 November 2019

M A Pedro

2

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1 November 2019

R C Scheffer

3

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1 November 2019

J H van Asseldonk

4

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1 November 2019

L C Souza

5

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1 November 2019

P L da Luz

6

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1 November 2019

O Augusto

7

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1 November 2019

F R Laurindo

8

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1 November 2019

publication date

1 November 2000

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Free Radical Biology and Medicine

1 November 2019

published in

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1 November 2019

volume

29

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1 November 2019

issue

9

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1 November 2019

page(s)

889-899

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NADH/NADPH Oxidase and Vascular Function

1 November 2019

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Increased NADH-Oxidase–Mediated Superoxide Production in the Early Stages of Atherosclerosis

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Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone

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p22phox is a critical component of the superoxide-generating NADH/NADPH oxidase system and regulates angiotensin II-induced hypertrophy in vascular smooth muscle cells

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The molybdoenzymes xanthine oxidase and aldehyde oxidase contain fast- and slow-DTNB reacting sulphydryl groups

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Inhibitors of the leukocyte superoxide generating oxidase: mechanisms of action and methods for their elucidation

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The cytochrome b and flavin content and properties of the O2- -forming NADPH oxidase solubilized from activated neutrophils

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Validation of lucigenin (bis-N-methylacridinium) as a chemilumigenic probe for detecting superoxide anion radical production by enzymatic and cellular systems

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Validation of lucigenin as a chemiluminescent probe to monitor vascular superoxide as well as basal vascular nitric oxide production

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7 January 2021

Lucigenin is a mediator of cytochrome C reduction but not of superoxide production

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Identifiers

DOI

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1 November 2019

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