Aggregation of ubiquitin and a mutant ALS-linked SOD1 protein correlate with disease progression and fragmentation of the Golgi apparatus (Q73450520)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10675580%20AND%20SRC:MED&resulttype=core&format=json

4 November 2019

title

Aggregation of ubiquitin and a mutant ALS-linked SOD1 protein correlate with disease progression and fragmentation of the Golgi apparatus (English)

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4 November 2019

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amyotrophic lateral sclerosis

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A Stieber

1

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4 November 2019

J O Gonatas

series ordinal

2

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4 November 2019

N K Gonatas

series ordinal

3

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4 November 2019

publication date

1 February 2000

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Journal of the Neurological Sciences

4 November 2019

published in

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4 November 2019

volume

173

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4 November 2019

page(s)

53-62

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4 November 2019

issue

1

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Amyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutase

4 November 2019

cites work

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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

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Genetics of amyotrophic lateral sclerosis

7 January 2021

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Evidence that all newly synthesized proteins destined for fast axonal transport pass through the Golgi apparatus

7 January 2021

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Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

7 January 2021

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The Golgi apparatus of spinal cord motor neurons in transgenic mice expressing mutant Cu,Zn superoxide dismutase becomes fragmented in early, preclinical stages of the disease

7 January 2021

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Do posttranslational modifications of CuZnSOD lead to sporadic amyotrophic lateral sclerosis?

7 January 2021

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Immunocytochemical visualization of the Golgi apparatus in several species, including human, and tissues with an antiserum against MG-160, a sialoglycoprotein of rat Golgi apparatus

7 January 2021

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The early stages of absorption of injected horseradish peroxidase in the proximal tubules of mouse kidney: ultrastructural cytochemistry by a new technique

7 January 2021

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A low expressor line of transgenic mice carrying a mutant human Cu,Zn superoxide dismutase (SOD1) gene develops pathological changes that most closely resemble those in human amyotrophic lateral sclerosis

7 January 2021

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The fragmented neuronal Golgi apparatus in amyotrophic lateral sclerosis includes the trans-Golgi-network: functional implications

7 January 2021

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The complete amino acid sequence of human Cu/Zn superoxide dismutase

7 January 2021

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Axonal transport of mutant superoxide dismutase 1 and focal axonal abnormalities in the proximal axons of transgenic mice.

7 January 2021

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Aggregation and Motor Neuron Toxicity of an ALS-Linked SOD1 Mutant Independent from Wild-Type SOD1

7 January 2021

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Nuclear and neuropil aggregates in Huntington's disease: relationship to neuropathology.

7 January 2021

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Speculating about spectrin: new insights into the Golgi-associated cytoskeleton

7 January 2021

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HISTOCHEMICAL AND ULTRASTRUCTURAL STUDIES ON HELA CELL CULTURES EXPOSED TO SPINDLE INHIBITORS WITH SPECIAL REFERENCE TO THE INTERPHASE CELL

7 January 2021

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The ubiquitin-proteasome proteolytic pathway

7 January 2021

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Lessons from the discovery of the ubiquitin system

7 January 2021

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A soluble ATP-dependent proteolytic system responsible for the degradation of abnormal proteins in reticulocytes

7 January 2021

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ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions.

7 January 2021

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Chaperoning brain diseases.

7 January 2021

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The ubiquitin pathway in Parkinson's disease.

7 January 2021

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10.1016/S0022-510X(99)00300-7

7 January 2021

Identifiers

DOI

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10675580%20AND%20SRC:MED&resulttype=core&format=json

4 November 2019

1 reference