Tandem-duplicated Flt3 constitutively activates STAT5 and MAP kinase and introduces autonomous cell growth in IL-3-dependent cell lines (Q73501441)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10698507%20AND%20SRC:MED&resulttype=core&format=json

4 November 2019

title

Tandem-duplicated Flt3 constitutively activates STAT5 and MAP kinase and introduces autonomous cell growth in IL-3-dependent cell lines (English)

1 reference

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4 November 2019

author name string

F Hayakawa

1

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4 November 2019

M Towatari

2

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4 November 2019

H Kiyoi

3

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4 November 2019

M Tanimoto

4

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4 November 2019

T Kitamura

5

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4 November 2019

H Saito

6

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4 November 2019

T Naoe

7

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4 November 2019

language of work or name

English

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publication date

1 February 2000

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4 November 2019

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4 November 2019

volume

19

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4 November 2019

issue

5

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4 November 2019

page(s)

624-631

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1203354

4 November 2019

exact match

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IL-3 dependent regulation of Bcl-xL gene expression by STAT5 in a bone marrow derived cell line

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Differential constitutive activation between STAT-related proteins and MAP kinase in primary acute myelogenous leukaemia.

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Tandem duplications of the FLT3 receptor gene are associated with leukemic transformation of myelodysplasia

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Internal tandem duplication of the FLT3 gene is a novel modality of elongation mutation which causes constitutive activation of the product

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FLT3 signaling in hematopoietic cells involves CBL, SHC and an unknown P115 as prominent tyrosine-phosphorylated substrates

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Targeted disruption of the flk2/flt3 gene leads to deficiencies in primitive hematopoietic progenitors

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Transcriptional regulation of the cyclin D1 promoter by STAT5: its involvement in cytokine-dependent growth of hematopoietic cells

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A receptor tyrosine kinase specific to hematopoietic stem and progenitor cell-enriched populations

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Mitogenic signalling and substrate specificity of the Flk2/Flt3 receptor tyrosine kinase in fibroblasts and interleukin 3-dependent hematopoietic cells

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Suppression of interleukin-3-induced gene expression by a C-terminal truncated Stat5: role of Stat5 in proliferation.

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Inhibition of MAP Kinase Kinase Blocks the Differentiation of PC-12 Cells Induced by Nerve Growth Factor

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Isolation and chromosomal localization of a novel FMS-like tyrosine kinase gene

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Fetal anemia and apoptosis of red cell progenitors in Stat5a-/-5b-/- mice: a direct role for Stat5 in Bcl-X(L) induction

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Constitutive activation of mitogen-activated protein kinase pathway in acute leukemia cells

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7 January 2021

Activation of Stat5 by platelet-derived growth factor (PDGF) is dependent on phosphorylation sites in PDGF beta-receptor juxtamembrane and kinase insert domains

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7 January 2021

Platelet-derived growth factor induces phosphorylation of multiple JAK family kinases and STAT proteins

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Mammary gland factor (MGF) is a novel member of the cytokine regulated transcription factor gene family and confers the prolactin response.

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Maximal activation of transcription by Stat1 and Stat3 requires both tyrosine and serine phosphorylation

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Internal tandem duplication of the FLT3 gene is preferentially seen in acute myeloid leukemia and myelodysplastic syndrome among various hematological malignancies. A study on a large series of patients and cell lines

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7 January 2021

Flt3 signaling involves tyrosyl-phosphorylation of SHP-2 and SHIP and their association with Grb2 and Shc in Baf3/Flt3 cells

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7 January 2021