Expression of matrix metalloproteinase-26 and tissue inhibitors of metalloproteinases TIMP-3 and -4 in benign endometrium and endometrial cancer (Q73507201)

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scientific article published on 01 June 2003

Language	Label	Description	Also known as
English	Expression of matrix metalloproteinase-26 and tissue inhibitors of metalloproteinases TIMP-3 and -4 in benign endometrium and endometrial cancer	scientific article published on 01 June 2003

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scholarly article

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4 November 2019

Expression of matrix metalloproteinase-26 and tissue inhibitors of metalloproteinases TIMP-3 and -4 in benign endometrium and endometrial cancer (English)

1 reference

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4 November 2019

endometrial cancer

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author name string

Renuka Tunuguntla

1

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4 November 2019

Daylene Ripley

2

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4 November 2019

Qing Xiang Amy Sang

3

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4 November 2019

Nasser Chegini

4

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4 November 2019

publication date

1 June 2003

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4 November 2019

Gynecologic Oncology

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4 November 2019

89

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4 November 2019

453-459

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4 November 2019

3

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4 November 2019

The many faces of metalloproteases: cell growth, invasion, angiogenesis and metastasis

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Complex roles of tissue inhibitors of metalloproteinases in cancer

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The role of matrix metalloproteinases in tumor angiogenesis and tumor metastasis

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Matrix metalloproteinases in angiogenesis: a moving target for therapeutic intervention

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Secretion of matrix metalloproteinases by human endometrial cells in vitro

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The matrix-degrading metalloproteinases

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Patterns of matrix metalloproteinase expression in cycling endometrium imply differential functions and regulation by steroid hormones

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Matrix metalloproteinases in normal menstruation

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Cancer statistics, 1999.

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Estrogen dependency in uterine endometrial cancers

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The association of transforming growth factor-beta 1 with myometrial invasion of endometrial carcinomas through effects on matrix metalloproteinase

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Enhanced production and activation of matrix metalloproteinase-7 (matrilysin) in human endometrial carcinomas

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Angiogenesis extent and expression of matrix metalloproteinase-2 and -9 correlate with upgrading and myometrial invasion in endometrial carcinoma

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Immunohistochemical studies on matrix metalloproteinase-9 (MMP-9) and type-IV collagen in endometrial carcinoma

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Identification and characterization of human endometase (Matrix metalloproteinase-26) from endometrial tumor

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Characterization of matrix metalloproteinase-26, a novel metalloproteinase widely expressed in cancer cells of epithelial origin

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Peptide substrate specificities and protein cleavage sites of human endometase/matrilysin-2/matrix metalloproteinase-26

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Cloning of MMP-26

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Differential expression of matrix metalloproteinase and tissue inhibitor of MMP in serosal tissue of intraperitoneal organs and adhesions

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Development and characterization of a new polyclonal antibody specifically against tissue inhibitor of metalloproteinases 4 in human breast cancer

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Expression of matrix metalloproteinase-26 and tissue inhibitor of metalloproteinase-4 in human normal cytotrophoblast cells and a choriocarcinoma cell line, JEG-3

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Endometrial progesterone receptors and markers of uterine receptivity in the window of implantation

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The role of matrix metalloproteinases and leukocytes in abnormal uterine bleeding associated with progestin-only contraceptives

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Circulating sex hormones and endometrial stromelysin-1 (matrix metalloproteinase-3) at the start of bleeding episodes in levonorgestrel-implant users

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Metalloproteinases and tissue inhibitor of metalloproteinase 1 (TIMP-1) in endometrial flushings from pre- and post-menopausal women and from women with endometrial adenocarcinoma

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Complex role of matrix metalloproteinases in angiogenesis

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Both tissue inhibitors of metalloproteinases-1 (TIMP-1) and TIMP-2 activate Ras but through different pathways

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Localization of MT1-MMP, TIMP-1, TIMP-2, and TIMP-3 messenger RNA in normal, hyperplastic, and neoplastic endometrium. Enhanced expression by endometrial adenocarcinomas is associated with low differentiation

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Identifiers

10.1016/S0090-8258(03)00077-5

1 reference

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4 November 2019

1 reference

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4 November 2019

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