Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing (Q73755804)
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scientific article published on 01 March 2001
| Language | Label | Description | Also known as |
|---|---|---|---|
| English | Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing |
scientific article published on 01 March 2001 |
Statements
Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation (English)
S Kato
K Nakashima
S Horiuchi
R Nagai
J Liu
A Hirano
M Takikawa
M Kato
I Nakano
S Sakoda
K Asayama
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