Upregulation of Meis1 and HoxA9 in acute lymphocytic leukemias with the t(4 : 11) abnormality (Q73787705)

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6 November 2019

title

Upregulation of Meis1 and HoxA9 in acute lymphocytic leukemias with the t(4 : 11) abnormality (English)

1 reference

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6 November 2019

author name string

T Rozovskaia

1

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6 November 2019

E Feinstein

2

1 reference

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6 November 2019

O Mor

3

1 reference

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6 November 2019

R Foa

4

1 reference

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6 November 2019

J Blechman

5

1 reference

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6 November 2019

T Nakamura

6

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6 November 2019

E Canaani

9

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6 November 2019

language of work or name

English

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publication date

1 February 2001

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6 November 2019

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6 November 2019

volume

20

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6 November 2019

issue

7

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6 November 2019

page(s)

874-878

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1204174

6 November 2019

exact match

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cites work

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The t(4;11) chromosome translocation of human acute leukemias fuses the ALL-1 gene, related to Drosophila trithorax, to the AF-4 gene

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Hoxa9 transforms primary bone marrow cells through specific collaboration with Meis1a but not Pbx1b

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Meis1, a PBX1-related homeobox gene involved in myeloid leukemia in BXH-2 mice

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Fusion of the nucleoporin gene NUP98 to HOXA9 by the chromosome translocation t(7;11)(p15;p15) in human myeloid leukaemia

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7 January 2021

Cooperative activation of Hoxa and Pbx1-related genes in murine myeloid leukaemias

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Acute myeloid leukemia in children treated with epipodophyllotoxins for acute lymphoblastic leukemia

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Trithorax and ASH1 interact directly and associate with the trithorax group-responsive bxd region of the Ultrabithorax promoter

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HoxA9-mediated immortalization of myeloid progenitors requires functional interactions with TALE cofactors Pbx and Meis

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HOXA9 forms triple complexes with PBX2 and MEIS1 in myeloid cells

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The oncogenic capacity of HRX-ENL requires the transcriptional transactivation activity of ENL and the DNA binding motifs of HRX.

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Overexpression of HOXA10 in murine hematopoietic cells perturbs both myeloid and lymphoid differentiation and leads to acute myeloid leukemia

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Trithorax- and Polycomb-group response elements within an Ultrabithorax transcription maintenance unit consist of closely situated but separable sequences

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Involvement of a homolog of Drosophila trithorax by 11q23 chromosomal translocations in acute leukemias

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