LEA-1/ICAM-1 interaction is essentially involved in the pathogenesis of delayed-type hypersensitivity-induced liver injury to picryl chloride (Q74497705)

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scientific article published on 01 January 1998

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English	LEA-1/ICAM-1 interaction is essentially involved in the pathogenesis of delayed-type hypersensitivity-induced liver injury to picryl chloride	scientific article published on 01 January 1998

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scholarly article

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11 November 2019

LEA-1/ICAM-1 interaction is essentially involved in the pathogenesis of delayed-type hypersensitivity-induced liver injury to picryl chloride (English)

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11 November 2019

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Q Xu

1

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11 November 2019

J Jiang

2

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11 November 2019

J Cao

3

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11 November 2019

F Wu

4

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11 November 2019

H Fujii

5

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11 November 2019

I Saiki

6

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1 January 1998

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11 November 2019

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11 November 2019

62

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11 November 2019

15

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11 November 2019

1281-1292

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11 November 2019

Hepatitis B virus immunopathogenesis

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Liver injury model in mice induced by a cellular immunologic mechanism--delayed-type hypersensitivity-induced liver injury to picryl chloride and phenotype of effector cell.

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One-shot delayed-type hypersensitivity reaction in the mouse liver causes a sustained liver injury to picryl chloride.

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The mechanism of biliary excretion of alpha 1-acid glycoprotein in the rat: evidence for a molecular weight-dependent, nonreceptor-mediated pathway

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Efficacy of intraportal injection of anti-ICAM-1 monoclonal antibody against liver cell injury following warm ischemia in the rat

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Effects of proinflammatory cytokines on cultivated primary human hepatocytes. Fluorometric measurement of intercellular adhesion molecule-1 and human leukocyte antigen-A, -B, -C, and -DR expression

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Inflammatory cytokines up-regulate intercellular adhesion molecule-1 expression on primary cultured mouse hepatocytes and T-lymphocyte adhesion

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The LFA-1/ICAM cell adhesion pathway is involved in tumor-cell lysis mediated by bispecific monoclonal-antibody-targeted T lymphocytes

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Purified intercellular adhesion molecule-1 (ICAM-1) is a ligand for lymphocyte function-associated antigen 1 (LFA-1)

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Functional evidence that intercellular adhesion molecule-1 (icam-1) is a ligand for lfa-1d-ependent adhesion in t cell-mediated cytotoxicity

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Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseases.

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Effect of injection of anti-CD4 and anti-CD8 monoclonal antibodies on the development of experimental systemic lupus erythematosus in mice

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7 January 2021

Identifiers

10.1016/S0024-3205(98)00062-9

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9566770%20AND%20SRC:MED&resulttype=core&format=json

11 November 2019

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9566770%20AND%20SRC:MED&resulttype=core&format=json

11 November 2019

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