Antioxidants protect against reactive oxygen species associated with adriamycin-treated cardiomyocytes (Q77352740)

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4 December 2019

title

Antioxidants protect against reactive oxygen species associated with adriamycin-treated cardiomyocytes (English)

1 reference

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4 December 2019

main subject

reactive oxygen species

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S M DeAtley

1

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4 December 2019

M Y Aksenov

2

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4 December 2019

M V Aksenova

3

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4 December 2019

B Harris

4

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4 December 2019

R Hadley

5

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4 December 2019

P Cole Harper

6

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4 December 2019

J M Carney

7

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4 December 2019

D A Butterfield

8

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4 December 2019

language of work or name

English

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publication date

1 February 1999

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4 December 2019

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4 December 2019

volume

136

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4 December 2019

issue

1

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4 December 2019

page(s)

41-46

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Anthracycline analogs: the past, present, and future

4 December 2019

cites work

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A clinicopathologic analysis of adriamycin cardiotoxicity

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Mechanism of adriamycin cardiotoxicity: evidence for oxidative stress

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Adriamycin induces protein oxidation in erythrocyte membranes

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Adriamycin: the role of lipid peroxidation in cardiac toxicity and tumor response

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Vitamin E deficiency accentuates adriamycin-induced cardiomyopathy and cell surface changes

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The effect of vitamin E and selenium on doxorubicin (Adriamycin) induced delayed toxicity in mice.

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Probucol protects against adriamycin cardiomyopathy without interfering with its antitumor effect

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Enalaprilat inhibits hydrogen peroxide production by murine mesangial cells exposed to high glucose concentrations

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Anti-inflammatory action of Pluchea sagittalis: Involvement of an antioxidant mechanism

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Direct evidence of oxidative injury produced by the Alzheimer's beta-amyloid peptide (1-40) in cultured hippocampal neurons

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7 January 2021

Analyses of the molecular mechanism of adriamycin-induced cardiotoxicity

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7 January 2021

Adriamycin-induced lipid peroxidation in mitochondria and microsomes

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7 January 2021

Oxidative and non-oxidative mechanisms in the inactivation of cardiac mitochondrial electron transport chain components by doxorubicin.

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7 January 2021

Cardiac mitochondrial calcium content during fatal doxorubicin toxicity

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7 January 2021

Disruption of mitochondrial calcium homeostasis following chronic doxorubicin administration.

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Superoxide, hydroxyl and fatty acid radical scavenging by aminosalicylates. Direct evaluation with electron spin resonance spectroscopy

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The anti-oxidant properties of 5-aminosalicylic acid

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7 January 2021

Prevention of ischemia/reperfusion-induced alterations in synaptosomal membrane-associated proteins and lipids by N-tert-butyl-alpha-phenylnitrone and difluoromethylornithine

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Prevention of hyperoxia-induced alterations in synaptosomal membrane-associated proteins by N-tert-butyl-alpha-phenylnitrone and 4-hydroxy-2,2,6,6-tetramethylpiperidin-1-oxyl (Tempol).

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7 January 2021

Free radical oxidation of brain proteins in accelerated senescence and its modulation by N-tert-butyl-alpha-phenylnitrone

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7 January 2021

10.1016/S0304-3835(98)00306-1

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10211937%20AND%20SRC:MED&resulttype=core&format=json

4 December 2019

1 reference