p57(Kip2) is degraded through the proteasome in osteoblasts stimulated to proliferation by transforming growth factor beta1 (Q77353495)

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4 December 2019

title

p57(Kip2) is degraded through the proteasome in osteoblasts stimulated to proliferation by transforming growth factor beta1 (English)

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4 December 2019

author name string

T Urano

1

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4 December 2019

H Yashiroda

2

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4 December 2019

M Muraoka

3

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4 December 2019

K Tanaka

4

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4 December 2019

T Hosoi

5

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4 December 2019

S Inoue

6

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4 December 2019

Y Ouchi

7

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H Toyoshima

8

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4 December 2019

language of work or name

English

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publication date

1 April 1999

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Journal of Biological Chemistry

4 December 2019

published in

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4 December 2019

volume

274

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4 December 2019

page(s)

12197-12200

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4 December 2019

issue

18

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Inhibitors of mammalian G1 cyclin-dependent kinases

4 December 2019

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Cloning of p57KIP2, a cyclin-dependent kinase inhibitor with unique domain structure and tissue distribution

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Interleukin-2-mediated elimination of the p27Kip1 cyclin-dependent kinase inhibitor prevented by rapamycin

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Role of the ubiquitin-proteasome pathway in regulating abundance of the cyclin-dependent kinase inhibitor p27

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Redistribution of the CDK inhibitor p27 between different cyclin.CDK complexes in the mouse fibroblast cell cycle and in cells arrested with lovastatin or ultraviolet irradiation

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Accumulation of the cyclin-dependent kinase inhibitor p27/Kip1 and the timing of oligodendrocyte differentiation

21 January 2018

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Altered cell differentiation and proliferation in mice lacking p57KIP2 indicates a role in Beckwith–Wiedemann syndrome

21 January 2018

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Ablation of the CDK inhibitor p57Kip2 results in increased apoptosis and delayed differentiation during mouse development.

21 January 2018

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Progressive development of the rat osteoblast phenotype in vitro: reciprocal relationships in expression of genes associated with osteoblast proliferation and differentiation during formation of the bone extracellular matrix.

21 January 2018

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21 January 2018

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21 January 2018

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Identifiers

DOI

10.1074/JBC.274.18.12197

1 reference

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4 December 2019

1 reference