Increased expression of constitutive nitric oxide synthase III, but not inducible nitric oxide synthase II, in human heart failure (Q77535879)

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6 December 2019

title

Increased expression of constitutive nitric oxide synthase III, but not inducible nitric oxide synthase II, in human heart failure (English)

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6 December 2019

author name string

B Stein

1

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6 December 2019

T Eschenhagen

2

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6 December 2019

J Rüdiger

3

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6 December 2019

H Scholz

4

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6 December 2019

U Förstermann

5

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6 December 2019

I Gath

6

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6 December 2019

language of work or name

English

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publication date

1 November 1998

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Journal of the American College of Cardiology

6 December 2019

published in

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6 December 2019

volume

32

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6 December 2019

issue

5

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6 December 2019

page(s)

1179-1186

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Nitric oxide synthase isozymes. Characterization, purification, molecular cloning, and functions.

6 December 2019

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Nitric oxide-dependent parasympathetic signaling is due to activation of constitutive endothelial (type III) nitric oxide synthase in cardiac myocytes

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Nitric oxide inhibits the positive inotropic response to beta-adrenergic stimulation in humans with left ventricular dysfunction

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Frequency-dependent activation of a constitutive nitric oxide synthase and regulation of contractile function in adult rat ventricular myocytes.

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Nitric oxide has no chronotropic effect in right atria isolated from rat heart.

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The ineffectiveness of the NO-cyclic GMP signaling pathway in the atrial myocardium

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Evidence against a role of nitric oxide in the indirect negative inotropic-effect of M-cholinoceptor stimulation in human ventricular myocardium.

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Decreased Catecholamine Sensitivity and β-Adrenergic-Receptor Density in Failing Human Hearts

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Altered myocardial force-frequency relation in human heart failure

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Is heart failure an abnormality of myocardial cell growth?

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Apoptosis in myocytes in end-stage heart failure

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Abnormal intracellular calcium handling in myocardium from patients with end-stage heart failure

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In vivo gene transfection of human endothelial cell nitric oxide synthase in cardiomyocytes causes apoptosis-like cell death. Identification using Sendai virus-coated liposomes

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Nitric oxide synthase activities in human myocardium

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Myocardial calcium-independent nitric oxide synthase activity is present in dilated cardiomyopathy, myocarditis, and postpartum cardiomyopathy but not in ischaemic or valvar heart disease

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7 January 2021

Expression of inducible nitric oxide synthase in human heart failure

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Tumour necrosis factor and inducible nitric oxide synthase in dilated cardiomyopathy

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Inducible nitric oxide synthase and tumor necrosis factor-alpha in myocardium in human dilated cardiomyopathy

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7 January 2021

Expression of inducible nitric oxide synthase in failing and non-failing human heart

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7 January 2021

Genetic expression of endothelial nitric oxide synthase in human atrial myocardium

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7 January 2021

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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Increased messenger RNA level of the inhibitory G protein alpha subunit Gi alpha-2 in human end-stage heart failure

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Identifiers

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6 December 2019

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