Cisplatin-induced nephrotoxicity is mediated by tumor necrosis factor-alpha produced by renal parenchymal cells (Q80066103)

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28 December 2019

title

Cisplatin-induced nephrotoxicity is mediated by tumor necrosis factor-alpha produced by renal parenchymal cells (English)

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28 December 2019

4

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28 December 2019

author name string

Zhang B

series ordinal

1

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28 December 2019

Ramesh G

series ordinal

2

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28 December 2019

Norbury CC

series ordinal

3

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28 December 2019

language of work or name

English

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publication date

28 March 2007

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28 December 2019

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28 December 2019

volume

72

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28 December 2019

issue

1

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28 December 2019

page(s)

37-44

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https://scigraph.springernature.com/pub.10.1038/sj.ki.5002242

28 December 2019

exact match

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cites work

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TNF-alpha mediates chemokine and cytokine expression and renal injury in cisplatin nephrotoxicity

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Interleukin-10 inhibits ischemic and cisplatin-induced acute renal injury

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Direct involvement of the receptor-mediated apoptotic pathways in cisplatin-induced renal tubular cell death

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Production of tumor necrosis factor by rat mesangial cells in response to bacterial lipopolysaccharide

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p38 MAP kinase inhibition ameliorates cisplatin nephrotoxicity in mice.

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Stimulated kidney tubular epithelial cells express membrane associated and secreted TNF alpha

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MHC class II, antigen presentation and tumor necrosis factor in renal tubular epithelial cells

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Nicotine induces endothelial TNF-alpha expression, which mediates growth retardation in vitro

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Naive monocytes can trigger transendothelial migration of peripheral blood cells through the induction of endothelial tumour necrosis factor-alpha

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Salicylate reduces cisplatin nephrotoxicity by inhibition of tumor necrosis factor-alpha.

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Acute nephrotoxic and obstructive injury primes the kidney to endotoxin-driven cytokine/chemokine production

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Antioxidant ameliorates cisplatin-induced renal tubular cell death through inhibition of death receptor-mediated pathways

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Role of angiotensin II-induced cAMP in mesangial TNF-alpha production

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7 January 2021

Parenchymal, But Not Leukocyte, TNF Receptor 2 Mediates T Cell-Dependent Hepatitis in Mice

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7 January 2021

Intrinsic renal cells are the major source of tumor necrosis factor contributing to renal injury in murine crescentic glomerulonephritis

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Renal protection from ischemia mediated by A2A adenosine receptors on bone marrow-derived cells

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7 January 2021

Platelet, but not endothelial, P-selectin is critical for neutrophil-mediated acute postischemic renal failure

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7 January 2021

A pathophysiologic role for T lymphocytes in murine acute cisplatin nephrotoxicity

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7 January 2021

Urinary actin, interleukin-6, and interleukin-8 may predict sustained arf after ischemic injury in renal allografts

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7 January 2021

Urine IL-18 is an early diagnostic marker for acute kidney injury and predicts mortality in the intensive care unit

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7 January 2021

Keratinocyte-derived chemokine is an early biomarker of ischemic acute kidney injury

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7 January 2021

Induction of p21WAF1/CIP1/SDI1 in kidney tubule cells affects the course of cisplatin-induced acute renal failure

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7 January 2021

Serial determination of glomerular filtration rate in conscious mice using FITC-inulin clearance

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7 January 2021

Antibody to intercellular adhesion molecule 1 protects the kidney against ischemic injury

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7 January 2021

Identifiers

DOI

10.1038/SJ.KI.5002242

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17396112%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

28 December 2019

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1 reference