Salt and stress synergize H. pylori-induced gastric lesions, cell proliferation, and p21 expression in Mongolian gerbils (Q80089385)

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28 December 2019

title

Salt and stress synergize H. pylori-induced gastric lesions, cell proliferation, and p21 expression in Mongolian gerbils (English)

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28 December 2019

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Meriones unguiculatus

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3

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28 December 2019

author name string

Armando Gamboa-Dominguez

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28 December 2019

Tom Ubbelohde

2

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28 December 2019

Luis Romano-Mazzoti

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Minerva Cervantes

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28 December 2019

Claudia Domínguez-Fonseca

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Maria de la Luz Estreber

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Guillermo M Ruíz-Palacios

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publication date

3 April 2007

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Digestive Diseases and Sciences

28 December 2019

published in

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28 December 2019

volume

52

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28 December 2019

page(s)

1517-1526

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28 December 2019

issue

6

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https://scigraph.springernature.com/pub.10.1007/s10620-006-9524-3

28 December 2019

exact match

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Helicobacter pylori infection enhances glandular stomach carcinogenesis in Mongolian gerbils treated with chemical carcinogens

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Helicobacter pylori infection and high dietary salt independently induce atrophic gastritis and intestinal metaplasia in commercially available outbred Mongolian gerbils.

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NF-kappaB- and C/EBPbeta-driven interleukin-1beta gene expression and PAK1-mediated caspase-1 activation play essential roles in interleukin-1beta release from Helicobacter pylori lipopolysaccharide-stimulated macrophages

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Attenuated apoptosis in H. pylori-colonized gastric mucosa of Mongolian gerbils in comparison with mice.

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12 December 2020

Influence of Helicobacter pylori infection on development of stress-induced gastric mucosal injury

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12 December 2020

Eradication of Helicobacter pylori induces apoptosis and inhibits proliferation of heterotopic proliferative glands in infected Mongolian gerbils

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