The human carcinoembryonic antigen (CEA) GPI anchor mediates anoikis inhibition by inactivation of the intrinsic death pathway (Q81343397)

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8 January 2020

title

The human carcinoembryonic antigen (CEA) GPI anchor mediates anoikis inhibition by inactivation of the intrinsic death pathway (English)

1 reference

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8 January 2020

author name string

Camacho-Leal P

series ordinal

1

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8 January 2020

Stanners CP

series ordinal

2

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8 January 2020

publication date

24 September 2007

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8 January 2020

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17891182%20AND%20SRC:MED&resulttype=core&format=json

8 January 2020

volume

27

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8 January 2020

issue

11

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8 January 2020

page(s)

1545-1553

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1210789

8 January 2020

exact match

0 references

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Carcinoembryonic antigen, a human tumor marker, functions as an intercellular adhesion molecule

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A co-clustering model involving alpha5beta1 integrin for the biological effects of GPI-anchored human carcinoembryonic antigen (CEA).

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HNF1 and/or HNF3 may contribute to the tissue specific expression of glucokinase gene.

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TRAIL-induced apoptosis requires Bax-dependent mitochondrial release of Smac/DIABLO.

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CEACAM6 gene silencing impairs anoikis resistance and in vivo metastatic ability of pancreatic adenocarcinoma cells

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Human carcinoembryonic antigen, an intercellular adhesion molecule, blocks fusion and differentiation of rat myoblasts

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Evidence for a function of death-receptor-related, death-domain-containing proteins in anoikis

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Integrins and anoikis

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Molecular mechanisms of "detachment-induced apoptosis--Anoikis".

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The carcinoembryonic antigen (CEA) family: structures, suggested functions and expression in normal and malignant tissues

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Deregulated expression of the human tumor marker CEA and CEA family member CEACAM6 disrupts tissue architecture and blocks colonocyte differentiation

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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FLICE/caspase-8 activation triggers anoikis induced by beta1-integrin blockade in human keratinocytes.

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Role of integrin-linked kinase in regulating phosphorylation of Akt and fibroblast survival in type I collagen matrices through a beta1 integrin viability signaling pathway

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Cell adhesion activity of non-specific cross-reacting antigen (NCA) and carcinoembryonic antigen (CEA) expressed on CHO cell surface: homophilic and heterophilic adhesion

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GPI-anchored CEA family glycoproteins CEA and CEACAM6 mediate their biological effects through enhanced integrin alpha5beta1-fibronectin interaction

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Clinging to life: cell to matrix adhesion and cell survival

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Anoikis and survival connections in the tumor microenvironment: is there a role in prostate cancer metastasis?

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Matrix detachment induces caspase-dependent cytochrome c release from mitochondria: inhibition by PKB/Akt but not Raf signalling

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The specificity for the differentiation blocking activity of carcinoembryonic antigen resides in its glycophosphatidyl-inositol anchor

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Carcinoembryonic antigen, a human tumor marker, cooperates with Myc and Bcl-2 in cellular transformation

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Get a ligand, get a life: integrins, signaling and cell survival

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The adhesion and differentiation-inhibitory activities of the immunoglobulin superfamily member, carcinoembryonic antigen, can be independently blocked

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beta 1 integrin regulates fibroblast viability during collagen matrix contraction through a phosphatidylinositol 3-kinase/Akt/protein kinase B signaling pathway

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Keratinocytes enriched for stem cells are protected from anoikis via an integrin signaling pathway in a Bcl-2 dependent manner.

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Caspase-9 activation results in downstream caspase-8 activation and bid cleavage in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced Parkinson's disease

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Early events in the anoikis program occur in the absence of caspase activation

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Retention of differentiation potentialities during prolonged cultivation of myogenic cells

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