Implication of dysregulation of the canonical wingless-type MMTV integration site (WNT) pathway in diabetic nephropathy (Q82126373)

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15 January 2020

title

Implication of dysregulation of the canonical wingless-type MMTV integration site (WNT) pathway in diabetic nephropathy (English)

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15 January 2020

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T Zhou

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X He

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R Cheng

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B Zhang

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R R Zhang

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Y Chen

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Y Takahashi

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A R Murray

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K Lee

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G Gao

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J-X Ma

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publication date

21 October 2011

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15 January 2020

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15 January 2020

volume

55

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issue

1

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15 January 2020

page(s)

255-266

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Mechanisms of tubulointerstitial injury in the kidney: final common pathways to end-stage renal failure

15 January 2020

cites work

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Interplay between Akt and p38 MAPK pathways in the regulation of renal tubular cell apoptosis associated with diabetic nephropathy

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Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice

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Specific down-regulation of connective tissue growth factor attenuates progression of nephropathy in mouse models of type 1 and type 2 diabetes

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Enhanced TGF-beta/Smad signaling in the early stage of diabetic nephropathy is independent of the AT1a receptor

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HNE-dependent molecular damage in diabetic nephropathy and its possible prevention by N-acetyl-cysteine and oxerutin

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CCN1/Cyr61 is regulated by the canonical Wnt signal and plays an important role in Wnt3A-induced osteoblast differentiation of mesenchymal stem cells

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12 December 2020

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12 December 2020

Role of oxidative stress in diabetic kidney disease.

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12 December 2020

Role of reactive oxygen species in the pathogenesis of diabetic nephropathy

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12 December 2020

The role of lipid peroxidation products and oxidative stress in activation of the canonical wingless-type MMTV integration site (WNT) pathway in a rat model of diabetic retinopathy

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12 December 2020

New insights into epithelial-mesenchymal transition in kidney fibrosis

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Salutary effect of pigment epithelium-derived factor in diabetic nephropathy: evidence for antifibrogenic activities

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Renal proximal tubular cell fibronectin accumulation in response to glucose is polyol pathway dependent

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