Loss of SOCS3 expression is associated with an increased risk of recurrent disease in breast carcinoma (Q82898508)

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18 January 2020

title

Loss of SOCS3 expression is associated with an increased risk of recurrent disease in breast carcinoma (English)

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18 January 2020

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Mingzhen Ying

1

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18 January 2020

Dawei Li

2

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18 January 2020

Linjun Yang

3

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18 January 2020

Mei Wang

4

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18 January 2020

Ning Wang

5

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18 January 2020

Ying Chen

6

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18 January 2020

Miaoxia He

7

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18 January 2020

Yajie Wang

8

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18 January 2020

publication date

14 February 2010

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Journal of Cancer Research and Clinical Oncology

18 January 2020

published in

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volume

136

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18 January 2020

issue

10

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page(s)

1617-1626

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https://scigraph.springernature.com/pub.10.1007/s00432-010-0819-6

18 January 2020

exact match

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cites work

Constitutive activation of Stat3 by the Src and JAK tyrosine kinases participates in growth regulation of human breast carcinoma cells

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12 December 2020

SOCS3 tyrosine phosphorylation as a potential bio-marker for myeloproliferative neoplasms associated with mutant JAK2 kinases

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Overexpression of SOCS3 is associated with decreased survival in a cohort of patients with de novo follicular lymphoma.

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Constitutive Stat3 activity up-regulates VEGF expression and tumor angiogenesis

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Positive and negative roles of IL-6, STAT3, and SOCS3 in inflammatory arthritis

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SOCS-1 participates in negative regulation of LPS responses

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Deletion of the SOCS3 gene in liver parenchymal cells promotes hepatitis-induced hepatocarcinogenesis.

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Placental defects and embryonic lethality in mice lacking suppressor of cytokine signaling 3

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12 December 2020

SOCS-3 is associated with vascular invasion and overall survival in hepatocellular carcinoma

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12 December 2020

SOCS3: an essential regulator of LIF receptor signaling in trophoblast giant cell differentiation

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12 December 2020

Leukemia inhibitory factor regulates trophoblast giant cell differentiation via Janus kinase 1-signal transducer and activator of transcription 3-suppressor of cytokine signaling 3 pathway.

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12 December 2020

Inhibition of growth and metastasis of human hepatocellular carcinoma by antisense oligonucleotide targeting signal transducer and activator of transcription 3.

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12 December 2020

Constitutive activation of Stat3 in fibroblasts transformed by diverse oncoproteins and in breast carcinoma cells.

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12 December 2020

SOCS3 as a tumor suppressor in breast cancer cells, and its regulation by PRL.

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12 December 2020

Negative regulators of cytokine signal transduction

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Suppressor of cytokine signaling 3 sensitizes anaplastic thyroid cancer to standard chemotherapy.

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The STATs of cancer--new molecular targets come of age

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SOCS-3 is frequently silenced by hypermethylation and suppresses cell growth in human lung cancer

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Methylation status of the SOCS3 gene in human malignant melanomas.

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Inhibition of Stat3 activation and tumor growth suppression of non-small cell lung cancer by G-quartet oligonucleotides.

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Sustained IL-6/STAT-3 signaling in cholangiocarcinoma cells due to SOCS-3 epigenetic silencing

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Stat3 promotes metastatic progression of prostate cancer.

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Prognostic relevance of SOCS3 hypermethylation in patients with glioblastoma multiforme.

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Decreased expression of SOCS-3 mRNA in breast cancer with lymph node metastasis

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Stat3-mediated Myc expression is required for Src transformation and PDGF-induced mitogenesis

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Roles of STAT3 in mediating the cell growth, differentiation and survival signals relayed through the IL-6 family of cytokine receptors

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12 December 2020

Regulation of interleukin-6-induced hepatic insulin resistance by mammalian target of rapamycin through the STAT3-SOCS3 pathway

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12 December 2020

Regulation of cytokine signaling by SOCS family molecules

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12 December 2020

Prognostic value of signal transducers and activators of transcription 3 in breast cancer

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12 December 2020

The evolution of lymph node assessment in breast cancer

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12 December 2020

Regulation of liver regeneration and hepatocarcinogenesis by suppressor of cytokine signaling 3

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12 December 2020

STAT3-mediated constitutive expression of SOCS3 in an undifferentiated rat trophoblast-like cell line

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12 December 2020

SOCS-3 is frequently methylated in head and neck squamous cell carcinoma and its precursor lesions and causes growth inhibition

1 reference

12 December 2020

10.1007/S00432-010-0819-6

Identifiers

DOI

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18 January 2020

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