Focal segmental glomerulosclerosis plays a major role in the progression of IgA nephropathy. I. Immunohistochemical studies (Q82900908)

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18 January 2020

title

Focal segmental glomerulosclerosis plays a major role in the progression of IgA nephropathy. I. Immunohistochemical studies (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21160460%20AND%20SRC:MED&resulttype=core&format=json

focal segmental glomerulosclerosis

18 January 2020

main subject

1 reference

1 reference

Gary S Hill

1

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18 January 2020

Khalil El Karoui

2

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18 January 2020

Alexandre Karras

3

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18 January 2020

Chantal Mandet

4

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18 January 2020

Jean-Paul Duong Van Huyen

5

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18 January 2020

Dominique Nochy

6

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18 January 2020

Patrick Bruneval

7

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18 January 2020

language of work or name

English

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publication date

15 December 2010

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18 January 2020

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18 January 2020

volume

79

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18 January 2020

issue

6

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18 January 2020

page(s)

635-642

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https://scigraph.springernature.com/pub.10.1038/ki.2010.466

18 January 2020

exact match

0 references

cites work

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The Oxford classification of IgA nephropathy: rationale, clinicopathological correlations, and classification

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Mesangial medium with IgA1 from IgA nephropathy inhibits nephrin expression in mouse podocytes.

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Effect of aggregated immunoglobulin A1 from immunoglobulin A nephropathy patients on nephrin expression in podocytes

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Aberrantly glycosylated IgA1 induces mesangial cells to produce platelet-activating factor that mediates nephrin loss in cultured podocytes

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Podocyte injury induced by mesangial-derived cytokines in IgA nephropathy

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Serum IgA1 from patients with IgA nephropathy up-regulates integrin-linked kinase synthesis and inhibits adhesive capacity in podocytes through indirect pathways.

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Activation of podocytes by mesangial-derived TNF-alpha: glomerulo-podocytic communication in IgA nephropathy

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Downregulation of Bcl-2 by podocytes is associated with progressive glomerular injury and clinical indices of poor renal prognosis in human IgA nephropathy

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Parietal podocytes in normal human glomeruli

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The parietal epithelial cell is crucially involved in human idiopathic focal segmental glomerulosclerosis

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Podocytes undergo phenotypic changes and express macrophagic-associated markers in idiopathic collapsing glomerulopathy

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Transdifferentiation of epithelial glomerular cells

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Significance of early phenotypic change of glomerular podocytes detected by Pax2 in primary focal segmental glomerulosclerosis

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Proliferating cells in HIV and pamidronate-associated collapsing focal segmental glomerulosclerosis are parietal epithelial cells

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Renal progenitor cells contribute to hyperplastic lesions of podocytopathies and crescentic glomerulonephritis

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Expression of nephrin in acquired human glomerular disease.

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The role of podocyte injury in the pathogenesis of focal segmental glomerulosclerosis

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7 January 2021

Activated parietal epithelial cells or dedifferentiated podocytes in FSGS: can we make the difference?

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Podocyte injury predicts prognosis in patients with iga nephropathy using a small amount of renal biopsy tissue

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