Effect of resveratrol on cartilage protection and apoptosis inhibition in experimental osteoarthritis of rabbit (Q83425376)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21327438%20AND%20SRC:MED&resulttype=core&format=json

22 January 2020

title

Effect of resveratrol on cartilage protection and apoptosis inhibition in experimental osteoarthritis of rabbit (English)

1 reference

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22 January 2020

1 reference

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Jing Wang

1

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22 January 2020

Jie-Sheng Gao

2

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22 January 2020

Jin-Wei Chen

3

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22 January 2020

Fen Li

4

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22 January 2020

Jing Tian

5

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22 January 2020

publication date

15 February 2011

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Rheumatology International

22 January 2020

published in

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22 January 2020

volume

32

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22 January 2020

page(s)

1541-1548

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22 January 2020

issue

6

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https://scigraph.springernature.com/pub.10.1007/s00296-010-1720-y

22 January 2020

exact match

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cites work

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Cytokines, tumor necrosis factor-alpha and interleukin-1beta, differentially regulate apoptosis in osteoarthritis cultured human chondrocytes.

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Resveratrol inhibits IL-1 beta-induced stimulation of caspase-3 and cleavage of PARP in human articular chondrocytes in vitro

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Effect of resveratrol in experimental osteoarthritis in rabbits

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Chemopreventive properties of trans-resveratrol are associated with inhibition of activation of the IkappaB kinase.

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Linkage of chondrocyte apoptosis and cartilage degradation in human osteoarthritis.

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Relationship of chondrocyte apoptosis to matrix degradation and swelling potential of osteoarthritic cartilage

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Resveratrol blocks interleukin-1beta-induced activation of the nuclear transcription factor NF-kappaB, inhibits proliferation, causes S-phase arrest, and induces apoptosis of acute myeloid leukemia cells

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Resveratrol as an anti-inflammatory and anti-aging agent: mechanisms and clinical implications

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The antioxidant resveratrol protects against chondrocyte apoptosis via effects on mitochondrial polarization and ATP production

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Resveratrol suppresses TNF-induced activation of nuclear transcription factors NF-kappa B, activator protein-1, and apoptosis: potential role of reactive oxygen intermediates and lipid peroxidation

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Osteoarthritis chondrocytes die by apoptosis: A possible pathway for osteoarthritis pathology

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Selective inhibition of inducible nitric oxide synthase reduces progression of experimental osteoarthritis in vivo: possible link with the reduction in chondrocyte apoptosis and caspase 3 level

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Selective inhibition of inducible nitric oxide synthase in experimental osteoarthritis is associated with reduction in tissue levels of catabolic factors

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Role of nitric oxide in the activation of NF-kappaB, AP-1 and NOS II expression in articular chondrocytes

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Caspase inhibitors reduce severity of cartilage lesions in experimental osteoarthritis

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Mitogen-activated protein kinase and nuclear factor kappaB together regulate interleukin-17-induced nitric oxide production in human osteoarthritic chondrocytes: possible role of transactivating factor mitogen-activated protein kinase-activated prot

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