Decrease in tetrahydrobiopterin as a possible cause of nephropathy in type II diabetic rats (Q83995811)

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30 January 2020

title

Decrease in tetrahydrobiopterin as a possible cause of nephropathy in type II diabetic rats (English)

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30 January 2020

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Okumura M

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Masada M

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Yoshida Y

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Shintaku H

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Hosoi M

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Okada N

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Konishi Y

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Morikawa T

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Miura K

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Imanishi M

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language of work or name

English

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publication date

14 June 2006

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30 January 2020

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30 January 2020

volume

70

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30 January 2020

page(s)

471-476

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issue

3

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https://scigraph.springernature.com/pub.10.1038/sj.ki.5000431

30 January 2020

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cites work

Nitric oxide in the kidney

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Renal nitric oxide production during the early phase of experimental diabetes mellitus

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Role of nitric oxide in diabetic nephropathy

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Structure-function aspects in the nitric oxide synthases

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Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension

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Abnormal biopterin metabolism is a major cause of impaired endothelium-dependent relaxation through nitric oxide/O2- imbalance in insulin-resistant rat aorta

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Impaired nitric oxide production in coronary endothelial cells of the spontaneously diabetic BB rat is due to tetrahydrobiopterin deficiency

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Tight blood pressure control and risk of macrovascular and microvascular complications in type 2 diabetes: UKPDS 38. UK Prospective Diabetes Study Group

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Glomerular hypertension as one cause of albuminuria in type II diabetic patients.

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Endothelial dysfunction

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Calcium channel blockade enhances nitric oxide synthase expression by cultured endothelial cells

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Benidipine stimulates nitric oxide synthase and improves coronary circulation in hypertensive rats

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A Ca channel blocker, benidipine, increases coronary blood flow and attenuates the severity of myocardial ischemia via NO-dependent mechanisms in dogs

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Spontaneous long-term hyperglycemic rat with diabetic complications. Otsuka Long-Evans Tokushima Fatty (OLETF) strain

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Role of nitrosative stress and peroxynitrite in the pathogenesis of diabetic complications. Emerging new therapeutical strategies

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Nephropathy in Zucker diabetic fat rat is associated with oxidative and nitrosative stress: prevention by chronic therapy with a peroxynitrite scavenger ebselen

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Review of alterations in endothelial nitric oxide production in diabetes: protective role of arginine on endothelial dysfunction

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Acute amelioration of diabetic endothelial dysfunction with a derivative of the nitric oxide synthase cofactor, tetrahydrobiopterin

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Oral administration of tetrahydrobiopterin prevents endothelial dysfunction and vascular oxidative stress in the aortas of insulin-resistant rats

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Tetrahydrobiopterin metabolism in the streptozotocin induced diabetic state in rats

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Renal disease in rats with type 2 diabetes is associated with decreased renal nitric oxide production

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Nitric oxide synthase inhibition in a spontaneously hypertensive rat model of diabetic nephropathy

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Tetrahydrobiopterin alters superoxide and nitric oxide release in prehypertensive rats

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The role of thiols in the apparent activation of rat brain nitric oxide synthase (NOS)

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Biopterin cofactor biosynthesis: independent regulation of GTP cyclohydrolase in adrenal medulla and cortex

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Up-regulation of GTP cyclohydrolase I and tetrahydrobiopterin by calcium influx.

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Long-acting calcium channel blocker benidipine suppresses expression of angiogenic growth factors and prevents cardiac remodelling in a Type II diabetic rat model

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Alteration of muscle fiber composition linking to insulin resistance and hypertension in fructose-fed rats

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