Tc9 cells, a new subset of CD8(+) T cells, support Th2-mediated airway inflammation (Q85741340)

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21 February 2020

title

Tc9 cells, a new subset of CD8(+) T cells, support Th2-mediated airway inflammation (English)

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21 February 2020

1

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21 February 2020

Tobias Bopp

11

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21 February 2020

Sebastian Reuter

12

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21 February 2020

author name string

Josephine Ritter

2

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21 February 2020

Tatjana Scholz

3

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21 February 2020

Lucia Campos

4

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21 February 2020

Anna Guralnik

5

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21 February 2020

Lucia Poncette

6

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21 February 2020

Hartmann Raifer

7

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21 February 2020

Stefanie Hagner

8

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21 February 2020

Holger Garn

9

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21 February 2020

Valerie Staudt

10

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21 February 2020

Christian Taube

13

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21 February 2020

Magdalena Huber

15

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publication date

31 January 2013

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European Journal of Immunology

21 February 2020

published in

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21 February 2020

volume

43

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21 February 2020

issue

3

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page(s)

606-618

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From interleukin-9 to T helper 9 cells.

21 February 2020

cites work

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7 January 2021

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Expression of interleukin 9 in the lungs of transgenic mice causes airway inflammation, mast cell hyperplasia, and bronchial hyperresponsiveness

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Interleukin-9 promotes allergen-induced eosinophilic inflammation and airway hyperresponsiveness in transgenic mice

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Transforming growth factor-beta 'reprograms' the differentiation of T helper 2 cells and promotes an interleukin 9-producing subset.

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IL-4 inhibits TGF-beta-induced Foxp3+ T cells and, together with TGF-beta, generates IL-9+ IL-10+ Foxp3(-) effector T cells

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7 January 2021

Interferon-regulatory factor 4 is essential for the developmental program of T helper 9 cells.

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The transcription factor PU.1 is required for the development of IL-9-producing T cells and allergic inflammation.

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The development of inflammatory T(H)-17 cells requires interferon-regulatory factor 4

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Dysregulated T helper cell differentiation in the absence of interferon regulatory factor 4.

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Interferon regulatory factor 4 (IRF4) interacts with NFATc2 to modulate interleukin 4 gene expression

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Transcription factor IRF4 determines germinal center formation through follicular T-helper cell differentiation

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STAT6-dependent regulation of Th9 development

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Regulation of IL-9 expression by IL-25 signaling.

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Segmental allergen challenge in patients with atopic asthma leads to increased IL-9 expression in bronchoalveolar lavage fluid lymphocytes

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IL-9 and its receptor in allergic and nonallergic lung disease: increased expression in asthma

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IL-9 induces VEGF secretion from human mast cells and IL-9/IL-9 receptor genes are overexpressed in atopic dermatitis

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7 January 2021

Heterogeneity of CD4(+) and CD8(+) T cells

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7 January 2021

Differential requirements for Runx proteins in CD4 repression and epigenetic silencing during T lymphocyte development.

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7 January 2021

Antigen-specific Th9 cells exhibit uniqueness in their kinetics of cytokine production and short retention at the inflammatory site

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7 January 2021

CD8+ T cell-mediated airway hyperresponsiveness and inflammation is dependent on CD4+IL-4+ T cells

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7 January 2021

A Th17-like developmental process leads to CD8(+) Tc17 cells with reduced cytotoxic activity

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Type 17 CD8+ T cells display enhanced antitumor immunity.

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IRF4 is essential for IL-21-mediated induction, amplification, and stabilization of the Th17 phenotype

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7 January 2021

Identifiers

DOI

10.1002/EJI.201242825

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21 February 2020

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