Pioglitazone inhibits angiotensin II-induced atrial fibroblasts proliferation via NF-κB/TGF-β1/TRIF/TRAF6 pathway (Q87649060)

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13 March 2020

title

Pioglitazone inhibits angiotensin II-induced atrial fibroblasts proliferation via NF-κB/TGF-β1/TRIF/TRAF6 pathway (English)

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13 March 2020

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Xiao-qing Chen

1

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13 March 2020

Xu Liu

2

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13 March 2020

Quan-xing Wang

3

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13 March 2020

Ming-jian Zhang

4

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13 March 2020

Meng Guo

5

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13 March 2020

Fang Liu

6

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13 March 2020

Wei-feng Jiang

7

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13 March 2020

Li Zhou

8

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13 March 2020

publication date

23 August 2014

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Experimental Cell Research

13 March 2020

published in

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13 March 2020

volume

330

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13 March 2020

page(s)

43-55

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13 March 2020

issue

1

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Atrial fibrillation pathophysiology: implications for management.

13 March 2020

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Pioglitazone attenuates inflammatory atrial fibrosis and vulnerability to atrial fibrillation induced by pressure overload in rats.

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PPAR-γ activator pioglitazone prevents age-related atrial fibrillation susceptibility by improving antioxidant capacity and reducing apoptosis in a rat model

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Pioglitazone, a peroxisome proliferator-activated receptor-gamma activator, attenuates atrial fibrosis and atrial fibrillation promotion in rabbits with congestive heart failure

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Pioglitazone ameliorates systolic and diastolic cardiac dysfunction in rat model of angiotensin II-induced hypertension

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Angiotensin II increases CTGF expression via MAPKs/TGF-β1/TRAF6 pathway in atrial fibroblasts

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IRF3 regulates cardiac fibrosis but not hypertrophy in mice during angiotensin II-induced hypertension

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Toll/IL-1 receptor domain-containing adaptor inducing IFN-beta (TRIF) associates with TNF receptor-associated factor 6 and TANK-binding kinase 1, and activates two distinct transcription factors, NF-kappa B and IFN-regulatory factor-3...

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Identifiers

DOI

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13 March 2020

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