Morphine inhibits doxorubicin-induced reactive oxygen species generation and nuclear factor kappaB transcriptional activation in neuroblastoma SH-SY5Y cells (Q95797465)

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scientific article published in 2007

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English	Morphine inhibits doxorubicin-induced reactive oxygen species generation and nuclear factor kappaB transcriptional activation in neuroblastoma SH-SY5Y cells	scientific article published in 2007

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scholarly article

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30 May 2020

Morphine inhibits doxorubicin-induced reactive oxygen species generation and nuclear factor kappaB transcriptional activation in neuroblastoma SH-SY5Y cells (English)

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30 May 2020

reactive oxygen species

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inferred from title

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based on heuristic

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author name string

Xin Lin

1

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30 May 2020

Qing Li

2

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30 May 2020

Yu-Jun Wang

3

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30 May 2020

Ya-Wen Ju

4

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30 May 2020

Zhi-Qiang Chi

5

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30 May 2020

Min-Wei Wang

6

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30 May 2020

Jing-Gen Liu

7

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30 May 2020

publication date

1 September 2007

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30 May 2020

number of pages

7

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Biochemical Journal

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30 May 2020

406

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30 May 2020

2

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30 May 2020

215-221

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30 May 2020

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As-needed morphine: yes, but at what dose and at what interval?

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Mechanism of apoptosis induced by doxorubicin through the generation of hydrogen peroxide.

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Carvedilol prevents doxorubicin-induced free radical release and apoptosis in cardiomyocytes in vitro

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Doxorubicin induces apoptosis in normal and tumor cells via distinctly different mechanisms. intermediacy of H(2)O(2)- and p53-dependent pathways

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10 February 2021

Evidence that activation of nuclear factor-kappaB is essential for the cytotoxic effects of doxorubicin and its analogues

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10 February 2021

Morphine suppresses lymphocyte apoptosis by blocking p53-mediated death signaling

1 reference

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10 February 2021

Reactive oxygen species mediate doxorubicin induced p53-independent apoptosis

1 reference

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10 February 2021

Role of reactive oxygen species and MAPKs in vanadate-induced G2/M phase arrest

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10 February 2021

Receptor activator of nuclear factor kappaB ligand plays a nonredundant role in doxorubicin-induced apoptosis

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10 February 2021

The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction.

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Morphine stimulates angiogenesis by activating proangiogenic and survival-promoting signaling and promotes breast tumor growth.

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Oxidative stress-mediated apoptosis. The anticancer effect of the sesquiterpene lactone parthenolide

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Activation of nuclear factor-kappaB during doxorubicin-induced apoptosis in endothelial cells and myocytes is pro-apoptotic: the role of hydrogen peroxide

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NF-kappa B activation mediates doxorubicin-induced cell death in N-type neuroblastoma cells

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Glutathione peroxidase-1 overexpression prevents ceramide production and partially inhibits apoptosis in doxorubicin-treated human breast carcinoma cells

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Protective effects of morphine in peroxynitrite-induced apoptosis of primary rat neonatal astrocytes: potential involvement of G protein and phosphatidylinositol 3-kinase (PI3 kinase).

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Morphine inhibits NF-kappaB nuclear binding in human neutrophils and monocytes by a nitric oxide-dependent mechanism

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Identifiers

10.1042/BJ20070186

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30 May 2020

PMC publication ID

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30 May 2020

PubMed publication ID

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30 May 2020

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