LEK1 is a potential inhibitor of pocket protein-mediated cellular processes (Q24297598)

RB transcriptional corepressor 1

GOA release 2020-03-11

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RB transcriptional corepressor like 2

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author name string

Mabelle Ashe

1

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Lil Pabon-Peña

2

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Ellen Dees

3

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Kristin L Price

4

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David Bader

Journal of Biological Chemistry

5

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language of work or name

English

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publication date

2 January 2004

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published in

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volume

279

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issue

1

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page(s)

664-76

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cites work

Differential regulation of the retinoblastoma family of proteins during cell proliferation and differentiation

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Reversal of terminal differentiation mediated by p107 in Rb-/- muscle cells

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The retinoblastoma gene family in differentiation and development.

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The retinoblastoma gene family is differentially expressed during embryogenesis.

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Genes in the RB pathway and their knockout in mice

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CENP-F is a protein of the nuclear matrix that assembles onto kinetochores at late G2 and is rapidly degraded after mitosis

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The C terminus of mitosin is essential for its nuclear localization, centromere/kinetochore targeting, and dimerization

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The cloning and analysis of LEK1 identifies variations in the LEK/centromere protein F/mitosin gene family

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Analysis of CMF1 reveals a bone morphogenetic protein-independent component of the cardiomyogenic pathway.

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The t(8;21) fusion protein interferes with AML-1B-dependent transcriptional activation

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Functional interaction between human topoisomerase IIalpha and retinoblastoma protein

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The retinoblastoma protein binds E2F residues required for activation in vivo and TBP binding in vitro

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Dual mechanisms for the inhibition of E2F binding to RB by cyclin-dependent kinase-mediated RB phosphorylation

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Acceleration of the G1/S phase transition by expression of cyclins D1 and E with an inducible system

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The E2F transcription factor is a cellular target for the RB protein

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Troglitazone inhibits growth of MCF-7 breast carcinoma cells by targeting G1 cell cycle regulators

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Searching new targets for anticancer drug design: the families of Ras and Rho GTPases and their effectors

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Transcriptional regulation. A closer look at E2F.

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Stable binding to E2F is not required for the retinoblastoma protein to activate transcription, promote differentiation, and suppress tumor cell growth

21 January 2018

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Retinoblastoma protein family in cell cycle and cancer: A review

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7 January 2021

p53-dependent and independent expression of p21 during cell growth, differentiation, and DNA damage

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Cyclin-dependent kinases and P53 pathways are activated independently and mediate Bax activation in neurons after DNA damage.

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New perspectives on retinoblastoma family functions in differentiation.

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Identifiers

DOI

10.1074/JBC.M308810200

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

14555653

1 reference

Consolidated OpenCitations Corpus – April 2017