The oxidative stressor arsenite activates vascular endothelial growth factor mRNA transcription by an ATF4-dependent mechanism (Q24298728)

positive regulation of transcription by RNA polymerase II

GOA release 2020-03-11

1 reference

Activating transcription factor 4

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

5

Steve F Abcouwer

Steve F

Abcouwer

1 reference

9 December 2023

C Nathaniel Roybal

1

C Nathaniel

Roybal

1 reference

9 December 2023

Lucy A Hunsaker

series ordinal

2

author given names

Lucy A

author last names

Hunsaker

1 reference

9 December 2023

Olena Barbash

series ordinal

3

author given names

Olena

author last names

Barbash

1 reference

9 December 2023

David L Vander Jagt

series ordinal

4

author given names

David L

author last names

Vander Jagt

1 reference

Journal of Biological Chemistry

9 December 2023

language of work or name

English

0 references

publication date

27 May 2005

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published in

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volume

280

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issue

21

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page(s)

20331-9

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Two distinct stress signaling pathways converge upon the CHOP promoter during the mammalian unfolded protein response

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ATF4 is a substrate of RSK2 and an essential regulator of osteoblast biology; implication for Coffin-Lowry Syndrome

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Regulated translation initiation controls stress-induced gene expression in mammalian cells

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Complexes containing activating transcription factor (ATF)/cAMP-responsive-element-binding protein (CREB) interact with the CCAAT/enhancer-binding protein (C/EBP)-ATF composite site to regulate Gadd153 expression during the stress response

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Distinct roles of activating transcription factor 6 (ATF6) and double-stranded RNA-activated protein kinase-like endoplasmic reticulum kinase (PERK) in transcription during the mammalian unfolded protein response

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Reinitiation involving upstream ORFs regulates ATF4 mRNA translation in mammalian cells

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Heme oxygenase is the major 32-kDa stress protein induced in human skin fibroblasts by UVA radiation, hydrogen peroxide, and sodium arsenite

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Evidence for a role of p38 kinase in hypoxia-inducible factor 1-independent induction of vascular endothelial growth factor expression by sodium arsenite

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Homocysteine increases the expression of vascular endothelial growth factor by a mechanism involving endoplasmic reticulum stress and transcription factor ATF4.

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Heme-regulated inhibitor kinase-mediated phosphorylation of eukaryotic translation initiation factor 2 inhibits translation, induces stress granule formation, and mediates survival upon arsenite exposure

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