Spinocerebellar ataxia type 35 (SCA35)-associated transglutaminase 6 mutants sensitize cells to apoptosis (Q24305167)

2

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Yu-Tao Liu

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Yan-Tao Ma

4

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Ying Zhou

5

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Hong Jiang

6

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Lu Shen

7

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Ji-Feng Guo

8

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Kun Xia

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Jia-Da Li

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Bei-Sha Tang

Biochemical and Biophysical Research Communications

11

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language of work or name

English

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publication date

11 January 2013

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published in

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volume

430

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issue

2

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page(s)

780-6

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cites work

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The mitochondrial pathways of apoptosis.

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TG2 transamidating activity acts as a reostat controlling the interplay between apoptosis and autophagy

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Mechanisms of cell death in polyglutamine expansion diseases

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Transglutaminase 2 in the balance of cell death and survival

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Caspase-8 is required for cell death induced by expanded polyglutamine repeats.

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Autosomal dominant cerebellar ataxias: polyglutamine expansions and beyond

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Apoptosis in neurodegenerative disorders

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Expanded polyglutamine in the Machado-Joseph disease protein induces cell death in vitro and in vivo

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Transglutaminase overexpression sensitizes neuronal cell lines to apoptosis by increasing mitochondrial membrane potential and cellular oxidative stress

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Distinct nuclear localization and activity of tissue transglutaminase

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Inhibition of "tissue" transglutaminase increases cell survival by preventing apoptosis

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Mutant protein kinase Cgamma found in spinocerebellar ataxia type 14 is susceptible to aggregation and causes cell death.

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The spinocerebellar ataxia 12 gene product and protein phosphatase 2A regulatory subunit Bbeta2 antagonizes neuronal survival by promoting mitochondrial fission

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TGM6 identified as a novel causative gene of spinocerebellar ataxias using exome sequencing

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Tissue transglutaminase protects epithelial ovarian cancer cells from cisplatin-induced apoptosis by promoting cell survival signaling

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Tissue transglutaminase serves as an inhibitor of apoptosis by cross-linking caspase 3 in thapsigargin-treated cells

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Genome-wide mRNA sequencing of a single canine cerebellar cortical degeneration case leads to the identification of a disease associated SPTBN2 mutation.

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Anti transglutaminase antibodies cause ataxia in mice

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Transglutaminase activation in neurodegenerative diseases.

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Tissue transglutaminase 2 promotes apoptosis of rat neonatal cardiomyocytes under oxidative stress

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Transglutaminase 2 inhibits apoptosis induced by calcium- overload through down-regulation of Bax

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Identifiers

DOI

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Fatcat ID

release_i6ksj7cfirh6dpp7odibfzn4ua

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/i6ksj7cfirh6dpp7odibfzn4ua

24 November 2022