IEX-1 directly interferes with RelA/p65 dependent transactivation and regulation of apoptosis (Q24306394)

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31 December 2019

title

IEX-1 directly interferes with RelA/p65 dependent transactivation and regulation of apoptosis (English)

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31 December 2019

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2

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31 December 2019

Alexander Arlt

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31 December 2019

8

Stefan Schreiber

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31 December 2019

author name string

Marie-Luise Kruse

3

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31 December 2019

Frauke Grohmann

4

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31 December 2019

Jörg Minkenberg

5

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31 December 2019

Neil D Perkins

6

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31 December 2019

Ulrich R Fölsch

7

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31 December 2019

Heiner Schäfer

9

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31 December 2019

language of work or name

English

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publication date

23 December 2007

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Biochimica et Biophysica Acta

31 December 2019

published in

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31 December 2019

volume

1783

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31 December 2019

issue

5

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31 December 2019

page(s)

941-952

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The putative apoptosis inhibitor IEX-1L is a mutant nonspliced variant of p22(PRG1/IEX-1) and is not expressed in vivo

31 December 2019

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B56-containing PP2A dephosphorylate ERK and their activity is controlled by the early gene IEX-1 and ERK

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Expression of the NF-kappa B target gene IEX-1 (p22/PRG1) does not prevent cell death but instead triggers apoptosis in Hela cells

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IEX-1, an immediate early gene, increases the rate of apoptosis in keratinocytes

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The early response gene IEX-1 attenuates NF-kappaB activation in 293 cells, a possible counter-regulatory process leading to enhanced cell death

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The Bcl-2 apoptotic switch in cancer development and therapy

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The apoptosis-inducing effect of gastrin on colorectal cancer cells relates to an increased IEX-1 expression mediating NF-kappa B inhibition

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Biomechanically Induced Gene iex-1 Inhibits Vascular Smooth Muscle Cell Proliferation and Neointima Formation

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Expression of the NF-kappa B target gene X-ray-inducible immediate early response factor-1 short enhances TNF-alpha-induced hepatocyte apoptosis by inhibiting Akt activation

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31 December 2019

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Consolidated OpenCitations Corpus – April 2017