Apolipoprotein CI inhibits scavenger receptor BI and increases plasma HDL levels in vivo (Q24312753)

phosphatidylcholine-sterol O-acyltransferase activator activity

GOA release 2020-03-11

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GOA release 2020-03-11

9

Louis M Havekes

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J Wouter Jukema

8

object named as

J Wouter Jukema

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Ko Willems van Dijk

5

object named as

Ko Willems van Dijk

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author name string

Willeke de Haan

1

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Ruud Out

2

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Jimmy F P Berbée

3

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Caroline C van der Hoogt

4

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Theo J C van Berkel

6

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Johannes A Romijn

7

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Patrick C N Rensen

Biochemical and Biophysical Research Communications

10

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language of work or name

English

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publication date

26 December 2008

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published in

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volume

377

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page(s)

1294-8

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issue

4

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cites work

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Apolipoprotein CI aggravates atherosclerosis development in ApoE-knockout mice despite mediating cholesterol efflux from macrophages

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Apolipoprotein CI causes hypertriglyceridemia independent of the very-low-density lipoprotein receptor and apolipoprotein CIII in mice

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Apolipoproteins C-II and C-III inhibit selective uptake of low- and high-density lipoprotein cholesteryl esters in HepG2 cells

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A targeted mutation in the murine gene encoding the high density lipoprotein (HDL) receptor scavenger receptor class B type I reveals its key role in HDL metabolism

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Mild dyslipidemia in mice following targeted inactivation of the hepatic lipase gene

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Human apolipoprotein C-I accounts for the ability of plasma high density lipoproteins to inhibit the cholesteryl ester transfer protein activity

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Scavenger receptor BI plays a role in facilitating chylomicron metabolism.

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Molecular mechanism of the blockade of plasma cholesteryl ester transfer protein by its physiological inhibitor apolipoprotein CI

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Plasma apolipoprotein CI protects against mortality from infection in old age.

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Apolipoprotein CI stimulates the response to lipopolysaccharide and reduces mortality in gram-negative sepsis

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Identification of scavenger receptor SR-BI as a high density lipoprotein receptor

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Fenofibrate reduces atherogenesis in ApoE*3Leiden mice: evidence for multiple antiatherogenic effects besides lowering plasma cholesterol

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Expression of human lecithin-cholesterol acyltransferase in transgenic mice. Effect of human apolipoprotein AI and human apolipoprotein all on plasma lipoprotein cholesterol metabolism

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In the absence of the low density lipoprotein receptor, human apolipoprotein C1 overexpression in transgenic mice inhibits the hepatic uptake of very low density lipoproteins via a receptor-associated protein-sensitive pathway

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Apolipoprotein C-I binds free fatty acids and reduces their intracellular esterification

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Fenofibrate increases HDL-cholesterol by reducing cholesteryl ester transfer protein expression

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Scavenger receptor BI facilitates the metabolism of VLDL lipoproteins in vivo

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10.1016/J.BBRC.2008.10.147

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Consolidated OpenCitations Corpus – April 2017

release_ypv3faljvjgwzjx55mwgozmytm

2622448

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https://api.fatcat.wiki/v0/release/ypv3faljvjgwzjx55mwgozmytm

24 November 2022

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2622448

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Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

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