BCL11A represses HBG transcription in K562 cells (Q24318302)

DNA-binding transcription repressor activity, RNA polymerase II-specific

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

Martin H Steinberg

3

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author name string

Zhiyi Chen

series ordinal

1

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Hong-yuan Luo

series ordinal

2

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David H K Chui

series ordinal

4

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language of work or name

English

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publication date

2009

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Blood Cells, Molecules and Diseases

PubMed

published in

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volume

42

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issue

2

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page(s)

144-9

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BCL11A is a major HbF quantitative trait locus in three different populations with beta-hemoglobinopathies

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FOG-1 recruits the NuRD repressor complex to mediate transcriptional repression by GATA-1.

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BCL11B functionally associates with the NuRD complex in T lymphocytes to repress targeted promoter

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Genome-wide association study shows BCL11A associated with persistent fetal hemoglobin and amelioration of the phenotype of beta-thalassemia

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A T-to-G transversion at nucleotide -567 upstream of HBG2 in a GATA-1 binding motif is associated with elevated hemoglobin F.

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Functional studies of BCL11A: characterization of the conserved BCL11A-XL splice variant and its interaction with BCL6 in nuclear paraspeckles of germinal center B cells

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