REDD1, a developmentally regulated transcriptional target of p63 and p53, links p63 to regulation of reactive oxygen species (Q24321795)

GOA release 2020-03-11

reactive oxygen species

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9

Daniel A Haber

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Leif W Ellisen

1

object named as

Leif W Ellisen

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author name string

Kate D Ramsayer

2

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Cory M Johannessen

3

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Annie Yang

4

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Hideyuki Beppu

5

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Karolina Minda

6

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Jonathan D Oliner

7

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Frank McKeon

Role of redox potential and reactive oxygen species in stress signaling

8

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language of work or name

English

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publication date

November 2002

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published in

Molecular Cell

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volume

10

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page(s)

995-1005

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issue

5

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cites work

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Telomere states and cell fates

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Patterning mechanisms controlling vertebrate limb development

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Mice lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control

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Human keratinocytes that express hTERT and also bypass a p16(INK4a)-enforced mechanism that limits life span become immortal yet retain normal growth and differentiation characteristics

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p63alpha and DeltaNp63alpha can induce cell cycle arrest and apoptosis and differentially regulate p53 target genes

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Regulation of p53 downstream genes.

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Definition of a consensus binding site for p53

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Cascades of transcriptional induction during human lymphocyte activation

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Redox-dependent signal transduction

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p63 and p73 are required for p53-dependent apoptosis in response to DNA damage

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A subset of tumor-derived mutant forms of p53 down-regulate p63 and p73 through a direct interaction with the p53 core domain

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The complexity of p53 modulation: emerging patterns from divergent signals

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The tyrosine kinase c-Abl regulates p73 in apoptotic response to cisplatin-induced DNA damage

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Transcription factors Oct-1 and NF-YA regulate the p53-independent induction of the GADD45 following DNA damage

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Regulation of p53 in response to DNA damage

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p53 regulates mitochondrial membrane potential through reactive oxygen species and induces cytochrome c-independent apoptosis blocked by Bcl-2.

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Reverse transcriptase motifs in the catalytic subunit of telomerase

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Expression monitoring by hybridization to high-density oligonucleotide arrays

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Reversible oxidation and inactivation of protein tyrosine phosphatases in vivo

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p63 is a p53 homologue required for limb and epidermal morphogenesis

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Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

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p53 and ATM: cell cycle, cell death, and cancer.

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Apaf-1 is a transcriptional target for E2F and p53

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Production of hydrogen peroxide by transforming growth factor-beta 1 and its involvement in induction of egr-1 in mouse osteoblastic cells

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Association of p63 with proliferative potential in normal and neoplastic human keratinocytes

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p63 identifies keratinocyte stem cells

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A model for p53-induced apoptosis

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Identification of a novel hypoxia-inducible factor 1-responsive gene, RTP801, involved in apoptosis.

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Wild-type p53 transactivates the KILLER/DR5 gene through an intronic sequence-specific DNA-binding site.

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A ribonucleotide reductase gene involved in a p53-dependent cell-cycle checkpoint for DNA damage

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Surfing the p53 network

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p63, a p53 homolog at 3q27-29, encodes multiple products with transactivating, death-inducing, and dominant-negative activities

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p63 is essential for regenerative proliferation in limb, craniofacial and epithelial development

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p73-deficient mice have neurological, pheromonal and inflammatory defects but lack spontaneous tumours

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p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage

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10.1016/S1097-2765(02)00706-2

Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

3450559

3450559

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Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

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