Characterization of human FAST-1, a TGF beta and activin signal transducer (Q24323087)

GOA release 2020-03-11

SMAD family member 2

1 reference

transcription factor binding

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

5

B Vogelstein

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Kenneth W. Kinzler

4

object named as

K W Kinzler

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author name string

S Zhou

1

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L Zawel

2

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C Lengauer

Activation of signalling by the activin receptor complex

3

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language of work or name

English

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publication date

July 1998

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published in

Molecular Cell

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volume

2

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issue

1

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page(s)

121-7

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cites work

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A transcriptional partner for MAD proteins in TGF-beta signalling

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Smad4 and FAST-1 in the assembly of activin-responsive factor

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Co-crystal structure of the HNF-3/fork head DNA-recognition motif resembles histone H5

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Rapid generation of chromosome-specific alphoid DNA probes using the polymerase chain reaction

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Transforming growth factor-beta signaling in epithelial cells

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TGF-beta signalling from cell membrane to nucleus through SMAD proteins

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Five years on the wings of fork head

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Drosophila Mad binds to DNA and directly mediates activation of vestigial by Decapentaplegic

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Regulation of translation in eukaryotic systems

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The TGF-beta family mediator Smad1 is phosphorylated directly and activated functionally by the BMP receptor kinase

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SMADs: mediators and regulators of TGF-beta signaling

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Partnership between DPC4 and SMAD proteins in TGF-beta signalling pathways.

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Genetic instability in colorectal cancers

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High-resolution mapping of human chromosome 11 by in situ hybridization with cosmid clones

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A human Mad protein acting as a BMP-regulated transcriptional activator

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Dual role of the Smad4/DPC4 tumor suppressor in TGFbeta-inducible transcriptional complexes

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MADR2 is a substrate of the TGFbeta receptor and its phosphorylation is required for nuclear accumulation and signaling

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Inactivation of the type II TGF-beta receptor in colon cancer cells with microsatellite instability

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TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4

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Identification of Smad2, a human Mad-related protein in the transforming growth factor beta signaling pathway

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A model for p53-induced apoptosis

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Caenorhabditis elegans genes sma-2, sma-3, and sma-4 define a conserved family of transforming growth factor beta pathway components

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Phosphorylation of Ser465 and Ser467 in the C terminus of Smad2 mediates interaction with Smad4 and is required for transforming growth factor-beta signaling

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DNA sequences mediating the transcriptional response of the Mix.2 homeobox gene to mesoderm induction.

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Demonstration that mutation of the type II transforming growth factor beta receptor inactivates its tumor suppressor activity in replication error-positive colon carcinoma cells

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Human Smad3 and Smad4 are sequence-specific transcription activators

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Targeted deletion of Smad4 shows it is required for transforming growth factor beta and activin signaling in colorectal cancer cells

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7 January 2021