Sublytic membrane-attack-complex (MAC) activation alters regulated rather than constitutive vascular endothelial growth factor (VEGF) secretion in retinal pigment epithelium monolayers (Q24598616)

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English	Sublytic membrane-attack-complex (MAC) activation alters regulated rather than constitutive vascular endothelial growth factor (VEGF) secretion in retinal pigment epithelium monolayers	scientific article

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scholarly article

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Sublytic membrane-attack-complex (MAC) activation alters regulated rather than constitutive vascular endothelial growth factor (VEGF) secretion in retinal pigment epithelium monolayers (English)

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2

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author name string

Kannan Kunchithapautham

1

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language of work or name

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publication date

8 July 2011

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Journal of Biological Chemistry

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286

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27

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23717-24

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Cell signals transduced by complement

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Inhibitors of vascular endothelial growth factor (VEGF) in the management of neovascular age-related macular degeneration: a review of current practice.

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Mitogen-activated protein kinase pathway mediates N-(4-hydroxyphenyl)retinamide-induced neuronal differentiation in the ARPE-19 human retinal pigment epithelial cell line

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VEGF modulation of retinal pigment epithelium resistance

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Ca2+ channels in retinal pigment epithelial cells regulate vascular endothelial growth factor secretion rates in health and disease.

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Membrane attack by complement

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Single channel currents induced by complement in antibody-coated cell membranes

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The Ras inhibitor farnesylthiosalicyclic acid (FTS) prevents nodule formation and development of preneoplastic foci of altered hepatocytes in rats.

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Anti-GM1 antibodies cause complement-mediated disruption of sodium channel clusters in peripheral motor nerve fibers.

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Complement activates the c-Jun N-terminal kinase/stress-activated protein kinase in glomerular epithelial cells

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Terminal complement complex C5b-9-treated human monocyte-derived dendritic cells undergo maturation and induce Th1 polarization.

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21 January 2018

Live cell imaging of outward and inward vesiculation induced by the complement c5b-9 complex.

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https://api.crossref.org/works/10.1074%2FJBC.M110.214593

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Glutamate-stimulated calcium activation of Ras/Erk pathway mediated by nitric oxide.

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21 January 2018

Age-related impairment of the transcriptional responses to oxidative stress in the mouse heart

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Novel mechanism for age-related macular degeneration: an equilibrium shift between the angiogenesis factors VEGF and PEDF.

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https://api.crossref.org/works/10.1074%2FJBC.M110.214593

21 January 2018

Vesicular removal by oligodendrocytes of membrane attack complexes formed by activated complement

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https://api.crossref.org/works/10.1074%2FJBC.M110.214593

21 January 2018

Calcium influx induces neurite growth through a Src-Ras signaling cassette

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https://pubmed.ncbi.nlm.nih.gov/21566137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Specific induction of intracellular calcium oscillations by complement membrane attack on oligodendroglia

1 reference

https://pubmed.ncbi.nlm.nih.gov/21566137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Incorporation of SP-40,40 into the soluble membrane attack complex (SMAC, SC5b-9) of complement

1 reference

https://pubmed.ncbi.nlm.nih.gov/21566137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The complement system and adaptive immunity

1 reference

https://pubmed.ncbi.nlm.nih.gov/21566137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Vascular endothelial growth factor and its receptors

1 reference

https://pubmed.ncbi.nlm.nih.gov/21566137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1074/JBC.M110.214593

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